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CTLA4-Ig reduces proliferation and inflammatory gene expression in muscle fibroblasts, corresponding to less fibrosis and inflammation in mdx muscular dystrophy.

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Title: CTLA4-Ig reduces proliferation and inflammatory gene expression in muscle fibroblasts, corresponding to less fibrosis and inflammation in mdx muscular dystrophy.
Authors: Wehling-Henricks M; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States., Kannan P; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Molecular, Cellular & Integrative Physiology Program, University of California, Los Angeles, California, United States., Thomas C; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Department of Anatomy, Cell Biology & Physiology, Indiana University School of Medicine, Indianapolis, Indiana, United States.; Indiana Center for Musculoskeletal Health, Indiana University School of Medicine, Indianapolis, Indiana, United States., Bal H; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States., Balu V; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States., Ochi E; Faculty of Bioscience and Applied Chemistry, Graduate School of Sports and Health Studies, Hosei University, Tokyo, Japan., Dorshkind K; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, University of California, Los Angeles, California, United States., Tidball JG; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Molecular, Cellular & Integrative Physiology Program, University of California, Los Angeles, California, United States.; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, University of California, Los Angeles, California, United States.
Source: American journal of physiology. Cell physiology [Am J Physiol Cell Physiol] 2026 May 01; Vol. 330 (5), pp. C1172-C1187. Date of Electronic Publication: 2026 Mar 19.
Publication Type: Journal Article
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901225 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1563 (Electronic) Linking ISSN: 03636143 NLM ISO Abbreviation: Am J Physiol Cell Physiol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: CTLA4-Ig reduces proliferation and inflammatory gene expression in muscle fibroblasts, corresponding to less fibrosis and inflammation in mdx muscular dystrophy.
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  Data: <searchLink fieldCode="AU" term="%22Wehling-Henricks+M%22">Wehling-Henricks M</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.<br /><searchLink fieldCode="AU" term="%22Kannan+P%22">Kannan P</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Molecular, Cellular & Integrative Physiology Program, University of California, Los Angeles, California, United States.<br /><searchLink fieldCode="AU" term="%22Thomas+C%22">Thomas C</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Department of Anatomy, Cell Biology & Physiology, Indiana University School of Medicine, Indianapolis, Indiana, United States.; Indiana Center for Musculoskeletal Health, Indiana University School of Medicine, Indianapolis, Indiana, United States.<br /><searchLink fieldCode="AU" term="%22Bal+H%22">Bal H</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.<br /><searchLink fieldCode="AU" term="%22Balu+V%22">Balu V</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.<br /><searchLink fieldCode="AU" term="%22Ochi+E%22">Ochi E</searchLink>; Faculty of Bioscience and Applied Chemistry, Graduate School of Sports and Health Studies, Hosei University, Tokyo, Japan.<br /><searchLink fieldCode="AU" term="%22Dorshkind+K%22">Dorshkind K</searchLink>; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, University of California, Los Angeles, California, United States.<br /><searchLink fieldCode="AU" term="%22Tidball+JG%22">Tidball JG</searchLink>; Department of Integrative Biology and Physiology, University of California, Los Angeles, California, United States.; Molecular, Cellular & Integrative Physiology Program, University of California, Los Angeles, California, United States.; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, University of California, Los Angeles, California, United States.
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  Data: <searchLink fieldCode="JN" term="%22100901225%22">American journal of physiology. Cell physiology</searchLink> [Am J Physiol Cell Physiol] 2026 May 01; Vol. 330 (5), pp. C1172-C1187. <i>Date of Electronic Publication: </i>2026 Mar 19.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901225 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1563 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203636143%22">03636143 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Cell Physiol <i>Subsets: </i>MEDLINE
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        Value: 10.1152/ajpcell.00860.2025
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