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GTPase Rab11b and effector Rab11-FIP2 promote NLRP3 stability during inflammasome priming.

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Title: GTPase Rab11b and effector Rab11-FIP2 promote NLRP3 stability during inflammasome priming.
Authors: Gravastrand CS; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Yurchenko M; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Kristensen S; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Skjesol A; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Chen C; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Ullmann S; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Iqbal Z; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Dahlen KR; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Rasheed K; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Nonstad U; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Ryan L; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway., Espevik T; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Clinic of Laboratory Medicine, St. Olavs Hospital, Trondheim, Norway., Husebye H; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway. harald.husebye@ntnu.no.
Source: The EMBO journal [EMBO J] 2026 May; Vol. 45 (9), pp. 2991-3021. Date of Electronic Publication: 2026 Mar 25.
Publication Type: Journal Article
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8208664 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1460-2075 (Electronic) Linking ISSN: 02614189 NLM ISO Abbreviation: EMBO J Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: GTPase Rab11b and effector Rab11-FIP2 promote NLRP3 stability during inflammasome priming.
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  Data: <searchLink fieldCode="AU" term="%22Gravastrand+CS%22">Gravastrand CS</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Yurchenko+M%22">Yurchenko M</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Kristensen+S%22">Kristensen S</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Skjesol+A%22">Skjesol A</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Chen+C%22">Chen C</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Ullmann+S%22">Ullmann S</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Iqbal+Z%22">Iqbal Z</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Dahlen+KR%22">Dahlen KR</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Rasheed+K%22">Rasheed K</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Nonstad+U%22">Nonstad U</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Ryan+L%22">Ryan L</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Espevik+T%22">Espevik T</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway.; Clinic of Laboratory Medicine, St. Olavs Hospital, Trondheim, Norway.<br /><searchLink fieldCode="AU" term="%22Husebye+H%22">Husebye H</searchLink>; Centre of Molecular Inflammation Research, Department of Clinical Molecular Medicine, Norwegian University of Science and Technology, Trondheim, Norway. harald.husebye@ntnu.no.
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