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Polystyrene nanoplastics induce hepatic steatosis by disrupting autophagic degradation of NCoR1 and suppressing PPARα-mediated fatty acid oxidation.

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Title: Polystyrene nanoplastics induce hepatic steatosis by disrupting autophagic degradation of NCoR1 and suppressing PPARα-mediated fatty acid oxidation.
Authors: Fang Y; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Wei T; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Zhang Z; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Li Y; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Yang M; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Chen H; Department of Nutrition and Food Hygiene, School of Public Health, Capital Medical University, Beijing, China., Wang Y; Department of Clinical Laboratory, Beijing Ditan Hospital, Capital Medical University, Beijing, 100015, China., Hao R; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Li Y; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Sun Z; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China., Wang J; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China. wangji@ccmu.edu.cn.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China. wangji@ccmu.edu.cn.
Source: Journal of nanobiotechnology [J Nanobiotechnology] 2026 Apr 02; Vol. 24 (1). Date of Electronic Publication: 2026 Apr 02.
Publication Type: Journal Article
Journal Info: Publisher: BioMed Central Country of Publication: England NLM ID: 101152208 Publication Model: Electronic Cited Medium: Internet ISSN: 1477-3155 (Electronic) Linking ISSN: 14773155 NLM ISO Abbreviation: J Nanobiotechnology Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Polystyrene nanoplastics induce hepatic steatosis by disrupting autophagic degradation of NCoR1 and suppressing PPARα-mediated fatty acid oxidation.
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  Data: <searchLink fieldCode="AU" term="%22Fang+Y%22">Fang Y</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Wei+T%22">Wei T</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Zhang+Z%22">Zhang Z</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Li+Y%22">Li Y</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Yang+M%22">Yang M</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Chen+H%22">Chen H</searchLink>; Department of Nutrition and Food Hygiene, School of Public Health, Capital Medical University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Wang+Y%22">Wang Y</searchLink>; Department of Clinical Laboratory, Beijing Ditan Hospital, Capital Medical University, Beijing, 100015, China.<br /><searchLink fieldCode="AU" term="%22Hao+R%22">Hao R</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Li+Y%22">Li Y</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Sun+Z%22">Sun Z</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China.<br /><searchLink fieldCode="AU" term="%22Wang+J%22">Wang J</searchLink>; Department of Toxicology and Sanitary Chemistry, School of Public Health, Capital Medical University, Beijing, 100069, China. wangji@ccmu.edu.cn.; Beijing Key Laboratory of Environmental and Aging, Capital Medical University, Beijing, 100069, China. wangji@ccmu.edu.cn.
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  Data: <searchLink fieldCode="JN" term="%22101152208%22">Journal of nanobiotechnology</searchLink> [J Nanobiotechnology] 2026 Apr 02; Vol. 24 (1). <i>Date of Electronic Publication: </i>2026 Apr 02.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22BioMed+Central%22">BioMed Central </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101152208 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1477-3155 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2214773155%22">14773155 </searchLink><i>NLM ISO Abbreviation: </i>J Nanobiotechnology <i>Subsets: </i>MEDLINE
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        Value: 10.1186/s12951-026-04304-6
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      – TitleFull: Polystyrene nanoplastics induce hepatic steatosis by disrupting autophagic degradation of NCoR1 and suppressing PPARα-mediated fatty acid oxidation.
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              Text: 2026 Apr 02
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