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Extracellular ATP promotes endocrine resistance in ER+ breast cancer through upregulation of PYGL.

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Title: Extracellular ATP promotes endocrine resistance in ER+ breast cancer through upregulation of PYGL.
Authors: Yu YQ; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Yu XY; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Li XF; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Cheng YF; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Zhou YT; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Mei F; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China., Weng L; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China. wengliang@pku.edu.cn., Tian XX; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China. tianxinxia@bjmu.edu.cn.
Source: Cell death & disease [Cell Death Dis] 2026 Apr 13; Vol. 17 (1). Date of Electronic Publication: 2026 Apr 13.
Publication Type: Journal Article
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Extracellular ATP promotes endocrine resistance in ER+ breast cancer through upregulation of PYGL.
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  Data: <searchLink fieldCode="AU" term="%22Yu+YQ%22">Yu YQ</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Yu+XY%22">Yu XY</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Li+XF%22">Li XF</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Cheng+YF%22">Cheng YF</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Zhou+YT%22">Zhou YT</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Mei+F%22">Mei F</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Weng+L%22">Weng L</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China. wengliang@pku.edu.cn.<br /><searchLink fieldCode="AU" term="%22Tian+XX%22">Tian XX</searchLink>; Department of Pathology, School of Basic Medical Sciences, Peking University Third Hospital, Peking University Health Science Center, Beijing, China. tianxinxia@bjmu.edu.cn.
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              Text: 2026 Apr 13
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