Selective Co‐activation of α7 and α4β2 Nicotinic Acetylcholine Receptors Reverses Beta‐amyloid‐induced Synaptic Dysfunction Jessica P. Roberts1,2,4, Sarah A. Stokoe1,2,4, Matheus F. Sathler2, Robert A. Nichols3, Seonil Kim1,21Molecular, Cellular and Integrative Neurosciences Program 2Department of Biomedical Sciences, Colorado State University, CO, 80523 3Department of Cell and Molecular Biology, University of Hawai'i at Manoa, Honolulu, HI, 96813 4These authors contribute equally

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Title: Selective Co‐activation of α7 and α4β2 Nicotinic Acetylcholine Receptors Reverses Beta‐amyloid‐induced Synaptic Dysfunction Jessica P. Roberts1,2,4, Sarah A. Stokoe1,2,4, Matheus F. Sathler2, Robert A. Nichols3, Seonil Kim1,21Molecular, Cellular and Integrative Neurosciences Program 2Department of Biomedical Sciences, Colorado State University, CO, 80523 3Department of Cell and Molecular Biology, University of Hawai'i at Manoa, Honolulu, HI, 96813 4These authors contribute equally
Authors: Roberts, Jessica
Source: FASEB journal: official publication of the Federation of American Societies for Experimental Biology May2021 Supplement 1; Vol. 35.
Journal Info: Publisher: Wiley-Blackwell ISSN: 0892-6638
Database: MEDLINE Ultimate
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  Data: Selective Co‐activation of α7 and α4β2 Nicotinic Acetylcholine Receptors Reverses Beta‐amyloid‐induced Synaptic Dysfunction Jessica P. Roberts1,2,4, Sarah A. Stokoe1,2,4, Matheus F. Sathler2, Robert A. Nichols3, Seonil Kim1,21Molecular, Cellular and Integrative Neurosciences Program 2Department of Biomedical Sciences, Colorado State University, CO, 80523 3Department of Cell and Molecular Biology, University of Hawai'i at Manoa, Honolulu, HI, 96813 4These authors contribute equally
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        Value: 10.1096/fasebj.2021.35.S1.03027
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      – TitleFull: Selective Co‐activation of α7 and α4β2 Nicotinic Acetylcholine Receptors Reverses Beta‐amyloid‐induced Synaptic Dysfunction Jessica P. Roberts1,2,4, Sarah A. Stokoe1,2,4, Matheus F. Sathler2, Robert A. Nichols3, Seonil Kim1,21Molecular, Cellular and Integrative Neurosciences Program 2Department of Biomedical Sciences, Colorado State University, CO, 80523 3Department of Cell and Molecular Biology, University of Hawai'i at Manoa, Honolulu, HI, 96813 4These authors contribute equally
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              Text: May2021 Supplement 1
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