Mechanisms of Cadmium Toxicity to Various Trophic Saltwater Organisms

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Title: Mechanisms of Cadmium Toxicity to Various Trophic Saltwater Organisms
Description: In this book, the authors summarize basic and experimental knowledge on sources and toxicity of cadmium and overview mechanisms of toxicity and detoxification for various trophic aquatic organisms exposed to cadmium different routes, which will assist ecological risk assessments involving cadmium toxicity.Cadmium is known to be both extremely toxic and ubiquitous in natural environments. It exists most commonly as a trace constituent in natural ecosystems, where its natural occurrence appears to not cause harm to the environment. However, it can be mobilized by a number of processes especially human activities. Anthropogenic sources of cadmium have contributed large amounts of this potentially toxic metal to the water cycle with ultimate impacts on aquatic ecosystems.
Authors: Wang, Zaosheng
Resource Type: eBook.
Subjects: Cadmium--Toxicology, Cadmium--Environmental aspects, Aquatic organisms--Effect of water pollution on
Categories: MEDICAL / Toxicology
Database: eBook Collection (EBSCOhost)
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  – Type: ebook-pdf
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  Availability: 0
Header DbId: nlebk
DbLabel: eBook Collection (EBSCOhost)
An: 410216
RelevancyScore: 1031
AccessLevel: 6
PubType: eBook
PubTypeId: ebook
PreciseRelevancyScore: 1031.17456054688
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Items – Name: Title
  Label: Title
  Group: Ti
  Data: Mechanisms of Cadmium Toxicity to Various Trophic Saltwater Organisms
– Name: Abstract
  Label: Description
  Group: Ab
  Data: In this book, the authors summarize basic and experimental knowledge on sources and toxicity of cadmium and overview mechanisms of toxicity and detoxification for various trophic aquatic organisms exposed to cadmium different routes, which will assist ecological risk assessments involving cadmium toxicity.Cadmium is known to be both extremely toxic and ubiquitous in natural environments. It exists most commonly as a trace constituent in natural ecosystems, where its natural occurrence appears to not cause harm to the environment. However, it can be mobilized by a number of processes especially human activities. Anthropogenic sources of cadmium have contributed large amounts of this potentially toxic metal to the water cycle with ultimate impacts on aquatic ecosystems.
– Name: Author
  Label: Authors
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  Data: <searchLink fieldCode="AR" term="%22Wang%2C+Zaosheng%22">Wang, Zaosheng</searchLink>
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  Data: eBook.
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  Data: <searchLink fieldCode="DE" term="%22Cadmium--Toxicology%22">Cadmium--Toxicology</searchLink><br /><searchLink fieldCode="DE" term="%22Cadmium--Environmental+aspects%22">Cadmium--Environmental aspects</searchLink><br /><searchLink fieldCode="DE" term="%22Aquatic+organisms--Effect+of+water+pollution+on%22">Aquatic organisms--Effect of water pollution on</searchLink>
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RecordInfo BibRecord:
  BibEntity:
    Classifications:
      – Code: 615.925662
        Scheme: ddc
        Type: prePub
    Languages:
      – Code: eng
        Text: English
    Subjects:
      – SubjectFull: Cadmium--Toxicology
        Type: general
      – SubjectFull: Cadmium--Environmental aspects
        Type: general
      – SubjectFull: Aquatic organisms--Effect of water pollution on
        Type: general
    Titles:
      – TitleFull: Mechanisms of Cadmium Toxicity to Various Trophic Saltwater Organisms
        Type: main
  BibRelationships:
    HasContributorRelationships:
      – PersonEntity:
          Name:
            NameFull: Wang, Zaosheng
      – PersonEntity:
          Name:
            NameFull: Wang, Zaosheng
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      – BibEntity:
          Dates:
            – D: 01
              M: 01
              Type: published
              Y: 2010
            – D: 04
              M: 02
              Type: profile
              Y: 2014
          Identifiers:
            – Type: isbn-print
              Value: 9781608766468
            – Type: isbn-electronic
              Value: 9781614702337
          Titles:
            – TitleFull: Mechanisms of Cadmium Toxicity to Various Trophic Saltwater Organisms
              Type: main
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