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Prenatal Risk and Infant Regulation: Indirect Pathways via Fetal Growth and Maternal Prenatal Stress and Anger.

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Title: Prenatal Risk and Infant Regulation: Indirect Pathways via Fetal Growth and Maternal Prenatal Stress and Anger.
Authors: Schuetze, Pamela, Eiden, Rina D., Colder, Craig R., Huestis, Marilyn A., Leonard, Kenneth E.
Source: Child Development. Mar/Apr2018, Vol. 89 Issue 2, pe123-e137. 15p. 1 Diagram, 3 Charts.
Subjects: Prenatal care, Fetal behavior, High-risk pregnancy, Mothers, Tobacco & health, Marijuana abuse, Physiological stress -- Risk factors, Anger, Substance abuse
Abstract: Pathways from maternal tobacco, marijuana, stress, and anger in pregnancy to infant reactivity and regulation (RR) at 9 months of infant age were examined in a low-income, diverse sample beginning in the first trimester of pregnancy, with fetal growth and postnatal stress/anger as potential mediators, and infant sex as a moderator. Participants were 247 dyads (173 substance-exposed infants). There were no direct effects of prenatal risk on RR and no moderation by sex. However, there were significant indirect effects on RR via poor fetal growth and higher postnatal anger. The study adds to the sparse literature on joint effects of tobacco and marijuana, and highlights the role of fetal growth and maternal anger as important pathways from prenatal risk to infant RR. [ABSTRACT FROM AUTHOR]
Copyright of Child Development is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
Database: Psychology and Behavioral Sciences Collection
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  Data: Prenatal Risk and Infant Regulation: Indirect Pathways via Fetal Growth and Maternal Prenatal Stress and Anger.
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  Data: <searchLink fieldCode="AR" term="%22Schuetze%2C+Pamela%22">Schuetze, Pamela</searchLink><br /><searchLink fieldCode="AR" term="%22Eiden%2C+Rina+D%2E%22">Eiden, Rina D.</searchLink><br /><searchLink fieldCode="AR" term="%22Colder%2C+Craig+R%2E%22">Colder, Craig R.</searchLink><br /><searchLink fieldCode="AR" term="%22Huestis%2C+Marilyn+A%2E%22">Huestis, Marilyn A.</searchLink><br /><searchLink fieldCode="AR" term="%22Leonard%2C+Kenneth+E%2E%22">Leonard, Kenneth E.</searchLink>
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  Data: <searchLink fieldCode="JN" term="%22Child+Development%22">Child Development</searchLink>. Mar/Apr2018, Vol. 89 Issue 2, pe123-e137. 15p. 1 Diagram, 3 Charts.
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  Data: <searchLink fieldCode="DE" term="%22Prenatal+care%22">Prenatal care</searchLink><br /><searchLink fieldCode="DE" term="%22Fetal+behavior%22">Fetal behavior</searchLink><br /><searchLink fieldCode="DE" term="%22High-risk+pregnancy%22">High-risk pregnancy</searchLink><br /><searchLink fieldCode="DE" term="%22Mothers%22">Mothers</searchLink><br /><searchLink fieldCode="DE" term="%22Tobacco+%26+health%22">Tobacco & health</searchLink><br /><searchLink fieldCode="DE" term="%22Marijuana+abuse%22">Marijuana abuse</searchLink><br /><searchLink fieldCode="DE" term="%22Physiological+stress+--+Risk+factors%22">Physiological stress -- Risk factors</searchLink><br /><searchLink fieldCode="DE" term="%22Anger%22">Anger</searchLink><br /><searchLink fieldCode="DE" term="%22Substance+abuse%22">Substance abuse</searchLink>
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  Label: Abstract
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  Data: Pathways from maternal tobacco, marijuana, stress, and anger in pregnancy to infant reactivity and regulation (RR) at 9 months of infant age were examined in a low-income, diverse sample beginning in the first trimester of pregnancy, with fetal growth and postnatal stress/anger as potential mediators, and infant sex as a moderator. Participants were 247 dyads (173 substance-exposed infants). There were no direct effects of prenatal risk on RR and no moderation by sex. However, there were significant indirect effects on RR via poor fetal growth and higher postnatal anger. The study adds to the sparse literature on joint effects of tobacco and marijuana, and highlights the role of fetal growth and maternal anger as important pathways from prenatal risk to infant RR. [ABSTRACT FROM AUTHOR]
– Name: AbstractSuppliedCopyright
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  Data: <i>Copyright of Child Development is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract.</i> (Copyright applies to all Abstracts.)
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RecordInfo BibRecord:
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      – Type: doi
        Value: 10.1111/cdev.12801
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      – Code: eng
        Text: English
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        PageCount: 15
        StartPage: e123
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      – SubjectFull: Prenatal care
        Type: general
      – SubjectFull: Fetal behavior
        Type: general
      – SubjectFull: High-risk pregnancy
        Type: general
      – SubjectFull: Mothers
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      – SubjectFull: Tobacco & health
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      – SubjectFull: Marijuana abuse
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      – SubjectFull: Physiological stress -- Risk factors
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      – SubjectFull: Anger
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      – SubjectFull: Substance abuse
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      – TitleFull: Prenatal Risk and Infant Regulation: Indirect Pathways via Fetal Growth and Maternal Prenatal Stress and Anger.
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              Text: Mar/Apr2018
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