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Sodium retention by uPA‐plasmin‐ENaC in nephrotic syndrome—Authors reply.

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Title: Sodium retention by uPA‐plasmin‐ENaC in nephrotic syndrome—Authors reply.
Authors: Hinrichs, Gitte R. (AUTHOR), Weyer, Kathrin (AUTHOR), Friis, Ulla G. (AUTHOR), Svenningsen, Per (AUTHOR), Lund, Ida Katrine (AUTHOR), Nielsen, Rikke (AUTHOR), Mollet, Géraldine (AUTHOR), Antignac, Corinne (AUTHOR), Bistrup, Claus (AUTHOR), Jensen, Boye L. (AUTHOR), Birn, Henrik (AUTHOR)
Source: Acta Physiologica. Apr2020, Vol. 228 Issue 4, p1-3. 3p.
Subjects: Nephrotic syndrome, Plasminogen activators, Serine proteinases, Brugada syndrome, Serum albumin
Abstract: Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome - Authors reply There is a great interest in exploring the mechanisms of oedema in nephrotic syndrome (NS) as evidenced by two parallel studies published in Acta Physiologica.[[1]] This has generated an editorial comment[3] which we respond to by this rebuttal. This seminal observation indicates that serine protease activity contributes to nephrotic sodium retention in mouse NS [8]; however, the importance of the individual protease(s) remains to be clarified. [Extracted from the article]
Copyright of Acta Physiologica is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
Database: Psychology and Behavioral Sciences Collection
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PubType: Academic Journal
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  Label: Title
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  Data: Sodium retention by uPA‐plasmin‐ENaC in nephrotic syndrome—Authors reply.
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  Data: <searchLink fieldCode="AR" term="%22Hinrichs%2C+Gitte+R%2E%22">Hinrichs, Gitte R.</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Weyer%2C+Kathrin%22">Weyer, Kathrin</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Friis%2C+Ulla+G%2E%22">Friis, Ulla G.</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Svenningsen%2C+Per%22">Svenningsen, Per</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Lund%2C+Ida+Katrine%22">Lund, Ida Katrine</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Nielsen%2C+Rikke%22">Nielsen, Rikke</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Mollet%2C+Géraldine%22">Mollet, Géraldine</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Antignac%2C+Corinne%22">Antignac, Corinne</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Bistrup%2C+Claus%22">Bistrup, Claus</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Jensen%2C+Boye+L%2E%22">Jensen, Boye L.</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Birn%2C+Henrik%22">Birn, Henrik</searchLink> (AUTHOR)
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  Data: <searchLink fieldCode="JN" term="%22Acta+Physiologica%22">Acta Physiologica</searchLink>. Apr2020, Vol. 228 Issue 4, p1-3. 3p.
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  Data: <searchLink fieldCode="DE" term="%22Nephrotic+syndrome%22">Nephrotic syndrome</searchLink><br /><searchLink fieldCode="DE" term="%22Plasminogen+activators%22">Plasminogen activators</searchLink><br /><searchLink fieldCode="DE" term="%22Serine+proteinases%22">Serine proteinases</searchLink><br /><searchLink fieldCode="DE" term="%22Brugada+syndrome%22">Brugada syndrome</searchLink><br /><searchLink fieldCode="DE" term="%22Serum+albumin%22">Serum albumin</searchLink>
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  Label: Abstract
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  Data: Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome - Authors reply There is a great interest in exploring the mechanisms of oedema in nephrotic syndrome (NS) as evidenced by two parallel studies published in Acta Physiologica.[[1]] This has generated an editorial comment[3] which we respond to by this rebuttal. This seminal observation indicates that serine protease activity contributes to nephrotic sodium retention in mouse NS [8]; however, the importance of the individual protease(s) remains to be clarified. [Extracted from the article]
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  Data: <i>Copyright of Acta Physiologica is the property of Wiley-Blackwell and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract.</i> (Copyright applies to all Abstracts.)
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              Text: Apr2020
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