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Late transience and persistence of contrast-induced encephalopathy: a case report and a literature update.

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Title: Late transience and persistence of contrast-induced encephalopathy: a case report and a literature update.
Authors: Antenucci, Pietro (AUTHOR), Antonioni, Annibale (AUTHOR), Calanca, Chiara (AUTHOR), Perin, Chiara (AUTHOR), Pugliatti, Maura (AUTHOR)
Source: Neurological Sciences. Jun2023, Vol. 44 Issue 6, p2187-2193. 7p. 2 Charts.
Subjects: Hyperperfusion, Lennox-Gastaut syndrome, Brain diseases, Posterior leukoencephalopathy syndrome, Symptoms, Positron emission tomography, Seizures (Medicine)
Abstract: Contrast-induced encephalopathy (CIE) is a rare and transient complication that occurs during or after angiographic procedures with intra-arterial administration of a contrast agent, especially iodine-based [[1]]. Characteristic computed tomography (CT) findings of CIE consist of transient cortical or subcortical enhancement, with a specific involvement of the affected lobes. Motor and sensory disturbances, cortical blindness, ophthalmoplegia, global aphasia and seizures are among CIE-described neurological signs and symptoms. In our case, EEG and PET findings were consistent with the neurotoxic hypothesis and no further radiological sings of ischemic lesions were found except the one in the left occipital lobe, so symptoms were related to contrast-induced neurotoxic damage. [Extracted from the article]
Copyright of Neurological Sciences is the property of Springer Nature and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
Database: Psychology and Behavioral Sciences Collection
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  Data: Late transience and persistence of contrast-induced encephalopathy: a case report and a literature update.
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  Data: <searchLink fieldCode="AR" term="%22Antenucci%2C+Pietro%22">Antenucci, Pietro</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Antonioni%2C+Annibale%22">Antonioni, Annibale</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Calanca%2C+Chiara%22">Calanca, Chiara</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Perin%2C+Chiara%22">Perin, Chiara</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Pugliatti%2C+Maura%22">Pugliatti, Maura</searchLink> (AUTHOR)
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  Data: <searchLink fieldCode="JN" term="%22Neurological+Sciences%22">Neurological Sciences</searchLink>. Jun2023, Vol. 44 Issue 6, p2187-2193. 7p. 2 Charts.
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  Data: <searchLink fieldCode="DE" term="%22Hyperperfusion%22">Hyperperfusion</searchLink><br /><searchLink fieldCode="DE" term="%22Lennox-Gastaut+syndrome%22">Lennox-Gastaut syndrome</searchLink><br /><searchLink fieldCode="DE" term="%22Brain+diseases%22">Brain diseases</searchLink><br /><searchLink fieldCode="DE" term="%22Posterior+leukoencephalopathy+syndrome%22">Posterior leukoencephalopathy syndrome</searchLink><br /><searchLink fieldCode="DE" term="%22Symptoms%22">Symptoms</searchLink><br /><searchLink fieldCode="DE" term="%22Positron+emission+tomography%22">Positron emission tomography</searchLink><br /><searchLink fieldCode="DE" term="%22Seizures+%28Medicine%29%22">Seizures (Medicine)</searchLink>
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  Data: Contrast-induced encephalopathy (CIE) is a rare and transient complication that occurs during or after angiographic procedures with intra-arterial administration of a contrast agent, especially iodine-based [[1]]. Characteristic computed tomography (CT) findings of CIE consist of transient cortical or subcortical enhancement, with a specific involvement of the affected lobes. Motor and sensory disturbances, cortical blindness, ophthalmoplegia, global aphasia and seizures are among CIE-described neurological signs and symptoms. In our case, EEG and PET findings were consistent with the neurotoxic hypothesis and no further radiological sings of ischemic lesions were found except the one in the left occipital lobe, so symptoms were related to contrast-induced neurotoxic damage. [Extracted from the article]
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  Data: <i>Copyright of Neurological Sciences is the property of Springer Nature and its content may not be copied or emailed to multiple sites without the copyright holder's express written permission. Additionally, content may not be used with any artificial intelligence tools or machine learning technologies. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract.</i> (Copyright applies to all Abstracts.)
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              Text: Jun2023
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