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Striatin knock out induces a gain of function of INa and impaired Ca2+ handling in mESC‐derived cardiomyocytes.

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Title: Striatin knock out induces a gain of function of INa and impaired Ca2+ handling in mESC‐derived cardiomyocytes.
Authors: Benzoni, P.1 (AUTHOR) patrizia.benzoni@unimi.it, Arici, M.2 (AUTHOR), Giannetti, F.1 (AUTHOR), Cospito, A.1 (AUTHOR), Prevostini, R.1 (AUTHOR), Volani, C.1,3 (AUTHOR), Fassina, L.4 (AUTHOR), Rosato‐Siri, M. D.3 (AUTHOR), Metallo, A.2 (AUTHOR), Gennaccaro, L.3 (AUTHOR), Suffredini, S.3 (AUTHOR), Foco, L.3 (AUTHOR), Mazzetti, S.5 (AUTHOR), Calogero, A.5 (AUTHOR), Cappelletti, G.5 (AUTHOR), Leibbrandt, A.6 (AUTHOR), Elling, U.6 (AUTHOR), Broso, F.3 (AUTHOR), Penninger, J. M.6,7 (AUTHOR), Pramstaller, P. P.3 (AUTHOR)
Source: Acta Physiologica. Aug2024, Vol. 240 Issue 8, p1-16.
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PubType: Academic Journal
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  Data: Striatin knock out induces a gain of function of I<subscript>Na</subscript> and impaired Ca<superscript>2+</superscript> handling in mESC‐derived cardiomyocytes.
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