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A glibenclamide-sensitive TRPM4-mediated component of CA1 excitatory postsynaptic potentials appears in experimental autoimmune encephalomyelitis.

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Bibliographic Details
Title: A glibenclamide-sensitive TRPM4-mediated component of CA1 excitatory postsynaptic potentials appears in experimental autoimmune encephalomyelitis.
Authors: Fearey, Brenna C.1,2 (AUTHOR), Binkle, Lars3 (AUTHOR), Mensching, Daniel3 (AUTHOR), Schulze, Christian1 (AUTHOR), Lohr, Christian4 (AUTHOR), Friese, Manuel A.3 (AUTHOR), Oertner, Thomas G.1 (AUTHOR), Gee, Christine E.1 (AUTHOR) christine.gee@zmnh.uni-hamburg.de
Source: Scientific Reports. 4/9/2022, Vol. 12 Issue 1, p1-12. 12p.
Database: Academic Search Ultimate
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