Senataxin helicase, the causal gene defect in ALS4, is a significant modifier of C9orf72 ALS G4C2 and arginine-containing dipeptide repeat toxicity.
Saved in:
| Title: | Senataxin helicase, the causal gene defect in ALS4, is a significant modifier of C9orf72 ALS G4C2 and arginine-containing dipeptide repeat toxicity. |
|---|---|
| Authors: | Bennett, Craig L.1,2 (AUTHOR), Dastidar, Somasish2,3 (AUTHOR), Arnold, Frederick J.1 (AUTHOR), McKinstry, Spencer U.2 (AUTHOR), Stockford, Cameron1 (AUTHOR), Freibaum, Brian D.4 (AUTHOR), Sopher, Bryce L.5 (AUTHOR), Wu, Meilin6 (AUTHOR), Seidner, Glen6 (AUTHOR), Joiner, William5 (AUTHOR), Taylor, J. Paul4,7 (AUTHOR), West, Ryan J. H.8,9 (AUTHOR) r.j.west@sheffield.ac.uk, La Spada, Albert R.1,2,10,11 (AUTHOR) alaspada@uci.edu |
| Source: | Acta Neuropathologica Communications. 10/17/2023, Vol. 11 Issue 1, p1-18. 18p. |
| Database: | Academic Search Ultimate |
|
Full text is not displayed to guests.
Login for full access.
|
|
Be the first to leave a comment!