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In Vivo Crispr Screening Identifies NF1/RASA1/TP53 CO-Mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis.

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Bibliographic Details
Title: In Vivo Crispr Screening Identifies NF1/RASA1/TP53 CO-Mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis.
Authors: Vu, Kenny1 (AUTHOR), Gunti, Sreenivasulu1 (AUTHOR), Viswanathan, Ramya1 (AUTHOR), Cho, Sungwoo1 (AUTHOR), Nandal, Anjali1 (AUTHOR), Larkin, Riley1 (AUTHOR), Zou, Jonathan1 (AUTHOR), Ramolia, Shivani1 (AUTHOR), Barbosa, Siani1 (AUTHOR), Gallia, Gary2 (AUTHOR), Rooper, Lisa2 (AUTHOR), Floudas, Charalampos1 (AUTHOR), Cheng, Hui3 (AUTHOR), Miller, Christine1 (AUTHOR), Guest, Mary1 (AUTHOR), Notaro, Marco4 (AUTHOR), Wu, Xiaolin1 (AUTHOR), Lechner, Matt5 (AUTHOR), Comoglio, Federico4 (AUTHOR), Edmonson, Elijah6 (AUTHOR)
Source: Journal of Neurological Surgery. Part B. Skull Base. 2025 Supplement 1, Vol. 86, pS1-S576. 576p.
Database: Academic Search Ultimate
Description
ISSN:21936331
DOI:10.1055/s-0045-1803258