In Vivo Crispr Screening Identifies NF1/RASA1/TP53 CO-Mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis.

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Title: In Vivo Crispr Screening Identifies NF1/RASA1/TP53 CO-Mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis.
Authors: Vu, Kenny1 (AUTHOR), Gunti, Sreenivasulu1 (AUTHOR), Viswanathan, Ramya1 (AUTHOR), Cho, Sungwoo1 (AUTHOR), Nandal, Anjali1 (AUTHOR), Larkin, Riley1 (AUTHOR), Zou, Jonathan1 (AUTHOR), Ramolia, Shivani1 (AUTHOR), Barbosa, Siani1 (AUTHOR), Gallia, Gary2 (AUTHOR), Rooper, Lisa2 (AUTHOR), Floudas, Charalampos1 (AUTHOR), Cheng, Hui3 (AUTHOR), Miller, Christine1 (AUTHOR), Guest, Mary1 (AUTHOR), Notaro, Marco4 (AUTHOR), Wu, Xiaolin1 (AUTHOR), Lechner, Matt5 (AUTHOR), Comoglio, Federico4 (AUTHOR), Edmonson, Elijah6 (AUTHOR)
Source: Journal of Neurological Surgery. Part B. Skull Base. 2025 Supplement 1, Vol. 86, pS1-S576. 576p.
Database: Academic Search Ultimate
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DbLabel: Academic Search Ultimate
An: 182992481
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PubTypeId: academicJournal
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  Data: <searchLink fieldCode="JN" term="%22Journal+of+Neurological+Surgery%2E+Part+B%2E+Skull+Base%22">Journal of Neurological Surgery. Part B. Skull Base</searchLink>. 2025 Supplement 1, Vol. 86, pS1-S576. 576p.
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=182992481
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        Value: 10.1055/s-0045-1803258
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        Text: English
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      – TitleFull: In Vivo Crispr Screening Identifies NF1/RASA1/TP53 CO-Mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis.
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              Text: 2025 Supplement 1
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