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Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling.

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Title: Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling.
Authors: Britschgi A; Friedrich Miescher Institute for Biomedical Research, CH-4058 Basel, Switzerland., Bill A, Brinkhaus H, Rothwell C, Clay I, Duss S, Rebhan M, Raman P, Guy CT, Wetzel K, George E, Popa MO, Lilley S, Choudhury H, Gosling M, Wang L, Fitzgerald S, Borawski J, Baffoe J, Labow M, Gaither LA, Bentires-Alj M
Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2013 Mar 12; Vol. 110 (11), pp. E1026-34. Date of Electronic Publication: 2013 Feb 19.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1091-6490 (Electronic) Linking ISSN: 00278424 NLM ISO Abbreviation: Proc Natl Acad Sci U S A Subsets: MEDLINE
Database: MEDLINE Ultimate
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PubType: Academic Journal
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  Data: Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling.
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  Data: <searchLink fieldCode="AU" term="%22Britschgi+A%22">Britschgi A</searchLink>; Friedrich Miescher Institute for Biomedical Research, CH-4058 Basel, Switzerland.<br /><searchLink fieldCode="AU" term="%22Bill+A%22">Bill A</searchLink><br /><searchLink fieldCode="AU" term="%22Brinkhaus+H%22">Brinkhaus H</searchLink><br /><searchLink fieldCode="AU" term="%22Rothwell+C%22">Rothwell C</searchLink><br /><searchLink fieldCode="AU" term="%22Clay+I%22">Clay I</searchLink><br /><searchLink fieldCode="AU" term="%22Duss+S%22">Duss S</searchLink><br /><searchLink fieldCode="AU" term="%22Rebhan+M%22">Rebhan M</searchLink><br /><searchLink fieldCode="AU" term="%22Raman+P%22">Raman P</searchLink><br /><searchLink fieldCode="AU" term="%22Guy+CT%22">Guy CT</searchLink><br /><searchLink fieldCode="AU" term="%22Wetzel+K%22">Wetzel K</searchLink><br /><searchLink fieldCode="AU" term="%22George+E%22">George E</searchLink><br /><searchLink fieldCode="AU" term="%22Popa+MO%22">Popa MO</searchLink><br /><searchLink fieldCode="AU" term="%22Lilley+S%22">Lilley S</searchLink><br /><searchLink fieldCode="AU" term="%22Choudhury+H%22">Choudhury H</searchLink><br /><searchLink fieldCode="AU" term="%22Gosling+M%22">Gosling M</searchLink><br /><searchLink fieldCode="AU" term="%22Wang+L%22">Wang L</searchLink><br /><searchLink fieldCode="AU" term="%22Fitzgerald+S%22">Fitzgerald S</searchLink><br /><searchLink fieldCode="AU" term="%22Borawski+J%22">Borawski J</searchLink><br /><searchLink fieldCode="AU" term="%22Baffoe+J%22">Baffoe J</searchLink><br /><searchLink fieldCode="AU" term="%22Labow+M%22">Labow M</searchLink><br /><searchLink fieldCode="AU" term="%22Gaither+LA%22">Gaither LA</searchLink><br /><searchLink fieldCode="AU" term="%22Bentires-Alj+M%22">Bentires-Alj M</searchLink>
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  Data: <searchLink fieldCode="JN" term="%227505876%22">Proceedings of the National Academy of Sciences of the United States of America</searchLink> [Proc Natl Acad Sci U S A] 2013 Mar 12; Vol. 110 (11), pp. E1026-34. <i>Date of Electronic Publication: </i>2013 Feb 19.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22National+Academy+of+Sciences%22">National Academy of Sciences </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>7505876 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1091-6490 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200278424%22">00278424 </searchLink><i>NLM ISO Abbreviation: </i>Proc Natl Acad Sci U S A <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=23431153
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              Text: 2013 Mar 12
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