Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1.

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Title: Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1.
Authors: Tursiella ML; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America., Bowman ER; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America., Wanzeck KC; Department of Biochemistry, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America., Throm RE; Department of Biochemistry, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America., Liao J; Department of Public Health Sciences, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America., Zhu J; Department of Public Health Sciences, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America., Sample CE; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.
Source: PLoS pathogens [PLoS Pathog] 2014 Oct 02; Vol. 10 (10), pp. e1004415. Date of Electronic Publication: 2014 Oct 02 (Print Publication: 2014).
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1.
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  Data: <searchLink fieldCode="AU" term="%22Tursiella+ML%22">Tursiella ML</searchLink>; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Bowman+ER%22">Bowman ER</searchLink>; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Wanzeck+KC%22">Wanzeck KC</searchLink>; Department of Biochemistry, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.<br /><searchLink fieldCode="AU" term="%22Throm+RE%22">Throm RE</searchLink>; Department of Biochemistry, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.<br /><searchLink fieldCode="AU" term="%22Liao+J%22">Liao J</searchLink>; Department of Public Health Sciences, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Zhu+J%22">Zhu J</searchLink>; Department of Public Health Sciences, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Sample+CE%22">Sample CE</searchLink>; Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, and the Penn State Hershey Cancer Institute, Hershey, Pennsylvania, United States of America.
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  Data: <searchLink fieldCode="JN" term="%22101238921%22">PLoS pathogens</searchLink> [PLoS Pathog] 2014 Oct 02; Vol. 10 (10), pp. e1004415. <i>Date of Electronic Publication: </i>2014 Oct 02 (<i>Print Publication: </i>2014).
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