Interleukin-1β enhances FasL-induced caspase-3/-7 activity without increasing apoptosis in primary mouse hepatocytes.

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Bibliographic Details
Title:	Interleukin-1β enhances FasL-induced caspase-3/-7 activity without increasing apoptosis in primary mouse hepatocytes.
Authors:	Lutz A; Department of Pharmaceutical Biology and Biotechnology, Albert Ludwigs University Freiburg, Freiburg, Germany., Sanwald J; Institute for System Dynamics, University of Stuttgart, Stuttgart, Germany., Thomas M; Dr. Margarete Fischer-Bosch Institute of Clinical Pharmacology, Stuttgart, Germany., Feuer R; Institute for System Dynamics, University of Stuttgart, Stuttgart, Germany., Sawodny O; Institute for System Dynamics, University of Stuttgart, Stuttgart, Germany., Ederer M; Institute for System Dynamics, University of Stuttgart, Stuttgart, Germany., Borner C; Institute of Molecular Medicine and Cell Research, Albert Ludwigs University Freiburg, Freiburg, Germany; Spemann Graduate School of Biology and Medicine (SGBM), Albert Ludwigs University Freiburg, Freiburg, Germany; Bioss - Centre for Biological Signaling Studies, Albert Ludwigs University Freiburg, Freiburg, Germany., Humar M; Department of Pharmaceutical Biology and Biotechnology, Albert Ludwigs University Freiburg, Freiburg, Germany., Merfort I; Department of Pharmaceutical Biology and Biotechnology, Albert Ludwigs University Freiburg, Freiburg, Germany; Spemann Graduate School of Biology and Medicine (SGBM), Albert Ludwigs University Freiburg, Freiburg, Germany.
Source:	PloS one [PLoS One] 2014 Dec 31; Vol. 9 (12), pp. e115603. Date of Electronic Publication: 2014 Dec 31 (Print Publication: 2014).
Publication Type:	Journal Article; Research Support, Non-U.S. Gov't
Journal Info:	Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 NLM ISO Abbreviation: PLoS One Subsets: MEDLINE
Database:	MEDLINE Ultimate

Description
ISSN:	1932-6203
DOI:	10.1371/journal.pone.0115603