Anabolism-Associated Mitochondrial Stasis Driving Lymphocyte Differentiation over Self-Renewal.

Saved in:
Bibliographic Details
Title: Anabolism-Associated Mitochondrial Stasis Driving Lymphocyte Differentiation over Self-Renewal.
Authors: Adams WC; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Chen YH; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Kratchmarov R; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Yen B; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Nish SA; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Lin WW; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Rothman NJ; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA., Luchsinger LL; Department of Medicine and Department of Microbiology and Immunology, Columbia University Medical Center, New York, NY 10032, USA., Klein U; Department of Pathology and Department of Microbiology and Immunology, Columbia University Medical Center, New York, NY 10032, USA., Busslinger M; Research Institute of Molecular Pathology, Vienna Biocenter, 1030 Vienna, Austria., Rathmell JC; Vanderbilt Centre for Immunobiology, Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, TN 37232, USA., Snoeck HW; Department of Medicine and Department of Microbiology and Immunology, Columbia University Medical Center, New York, NY 10032, USA., Reiner SL; Department of Microbiology and Immunology and Department of Pediatrics, Columbia University Medical Center, New York, NY 10032, USA. Electronic address: sr2978@cumc.columbia.edu.
Source: Cell reports [Cell Rep] 2016 Dec 20; Vol. 17 (12), pp. 3142-3152.
Publication Type: Journal Article
Journal Info: Publisher: Cell Press Country of Publication: United States NLM ID: 101573691 Publication Model: Print Cited Medium: Internet ISSN: 2211-1247 (Electronic) NLM ISO Abbreviation: Cell Rep Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2211-1247
DOI:10.1016/j.celrep.2016.11.065