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Hypoxia-inducible factor prolyl-4-hydroxylation in FOXD1 lineage cells is essential for normal kidney development.

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Title: Hypoxia-inducible factor prolyl-4-hydroxylation in FOXD1 lineage cells is essential for normal kidney development.
Authors: Kobayashi H; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA., Liu J; Section of Pediatric Nephrology, Department of Pediatrics, Tulane University Health Sciences Center, New Orleans, Louisiana, USA; The Hypertension and Renal Centers of Excellence, Tulane University Health Sciences Center, New Orleans, Louisiana, USA., Urrutia AA; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA., Burmakin M; Division of Vascular Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Stockholm, Sweden., Ishii K; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA., Rajan M; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA., Davidoff O; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA., Saifudeen Z; Section of Pediatric Nephrology, Department of Pediatrics, Tulane University Health Sciences Center, New Orleans, Louisiana, USA; The Hypertension and Renal Centers of Excellence, Tulane University Health Sciences Center, New Orleans, Louisiana, USA., Haase VH; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA; Department of Cancer Biology and Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA. Electronic address: volker.haase@vanderbilt.edu.
Source: Kidney international [Kidney Int] 2017 Dec; Vol. 92 (6), pp. 1370-1383. Date of Electronic Publication: 2017 Aug 26.
Publication Type: Journal Article
Journal Info: Publisher: Elsevier Country of Publication: United States NLM ID: 0323470 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1523-1755 (Electronic) Linking ISSN: 00852538 NLM ISO Abbreviation: Kidney Int Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Hypoxia-inducible factor prolyl-4-hydroxylation in FOXD1 lineage cells is essential for normal kidney development.
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  Data: <searchLink fieldCode="AU" term="%22Kobayashi+H%22">Kobayashi H</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA.<br /><searchLink fieldCode="AU" term="%22Liu+J%22">Liu J</searchLink>; Section of Pediatric Nephrology, Department of Pediatrics, Tulane University Health Sciences Center, New Orleans, Louisiana, USA; The Hypertension and Renal Centers of Excellence, Tulane University Health Sciences Center, New Orleans, Louisiana, USA.<br /><searchLink fieldCode="AU" term="%22Urrutia+AA%22">Urrutia AA</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA.<br /><searchLink fieldCode="AU" term="%22Burmakin+M%22">Burmakin M</searchLink>; Division of Vascular Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Stockholm, Sweden.<br /><searchLink fieldCode="AU" term="%22Ishii+K%22">Ishii K</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA.<br /><searchLink fieldCode="AU" term="%22Rajan+M%22">Rajan M</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA.<br /><searchLink fieldCode="AU" term="%22Davidoff+O%22">Davidoff O</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA.<br /><searchLink fieldCode="AU" term="%22Saifudeen+Z%22">Saifudeen Z</searchLink>; Section of Pediatric Nephrology, Department of Pediatrics, Tulane University Health Sciences Center, New Orleans, Louisiana, USA; The Hypertension and Renal Centers of Excellence, Tulane University Health Sciences Center, New Orleans, Louisiana, USA.<br /><searchLink fieldCode="AU" term="%22Haase+VH%22">Haase VH</searchLink>; Department of Medicine, Vanderbilt University School of Medicine, Nashville, Tennessee, USA; Medical and Research Services, Department of Veterans Affairs Hospital, Tennessee Valley Healthcare System, Nashville, Tennessee, USA; Department of Cancer Biology and Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA. Electronic address: volker.haase@vanderbilt.edu.
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  Data: <searchLink fieldCode="JN" term="%220323470%22">Kidney international</searchLink> [Kidney Int] 2017 Dec; Vol. 92 (6), pp. 1370-1383. <i>Date of Electronic Publication: </i>2017 Aug 26.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier%22">Elsevier </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>0323470 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1523-1755 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200852538%22">00852538 </searchLink><i>NLM ISO Abbreviation: </i>Kidney Int <i>Subsets: </i>MEDLINE
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        Value: 10.1016/j.kint.2017.06.015
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              Text: 2017 Dec
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