Angiopoietin-like 4 Is a Wnt Signaling Antagonist that Promotes LRP6 Turnover.

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Title: Angiopoietin-like 4 Is a Wnt Signaling Antagonist that Promotes LRP6 Turnover.
Authors: Kirsch N; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany., Chang LS; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany., Koch S; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany., Glinka A; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany., Dolde C; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany., Colozza G; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA., Benitez MDJ; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA., De Robertis EM; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA., Niehrs C; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany; Institute of Molecular Biology (IMB), 55128 Mainz, Germany. Electronic address: niehrs@dkfz-heidelberg.de.
Source: Developmental cell [Dev Cell] 2017 Oct 09; Vol. 43 (1), pp. 71-82.e6.
Publication Type: Journal Article
Journal Info: Publisher: Cell Press Country of Publication: United States NLM ID: 101120028 Publication Model: Print Cited Medium: Internet ISSN: 1878-1551 (Electronic) Linking ISSN: 15345807 NLM ISO Abbreviation: Dev Cell Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Angiopoietin-like 4 Is a Wnt Signaling Antagonist that Promotes LRP6 Turnover.
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  Data: <searchLink fieldCode="AU" term="%22Kirsch+N%22">Kirsch N</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Chang+LS%22">Chang LS</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Koch+S%22">Koch S</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Glinka+A%22">Glinka A</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Dolde+C%22">Dolde C</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Colozza+G%22">Colozza G</searchLink>; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA.<br /><searchLink fieldCode="AU" term="%22Benitez+MDJ%22">Benitez MDJ</searchLink>; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA.<br /><searchLink fieldCode="AU" term="%22De+Robertis+EM%22">De Robertis EM</searchLink>; Howard Hughes Medical Institute and Department of Biological Chemistry, University of California, Los Angeles, CA 90095-1662, USA.<br /><searchLink fieldCode="AU" term="%22Niehrs+C%22">Niehrs C</searchLink>; Division of Molecular Embryology, DKFZ-ZMBH Alliance, Deutsches Krebsforschungszentrum (DKFZ), 69120 Heidelberg, Germany; Institute of Molecular Biology (IMB), 55128 Mainz, Germany. Electronic address: niehrs@dkfz-heidelberg.de.
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  Data: <searchLink fieldCode="JN" term="%22101120028%22">Developmental cell</searchLink> [Dev Cell] 2017 Oct 09; Vol. 43 (1), pp. 71-82.e6.
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        Value: 10.1016/j.devcel.2017.09.011
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              Text: 2017 Oct 09
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