TorsinA dysfunction causes persistent neuronal nuclear pore defects.
Saved in:
| Title: | TorsinA dysfunction causes persistent neuronal nuclear pore defects. |
|---|---|
| Authors: | Pappas SS; Department of Neurology., Liang CC; Department of Neurology., Kim S; Cellular and Molecular Biology Program., Rivera CO; Department of Neurology., Dauer WT; Department of Neurology.; Cellular and Molecular Biology Program.; Department of Cell and Developmental Biology.; VA Ann Arbor Health System, University of Michigan Medical School, Ann Arbor, MI 48109, USA. |
| Source: | Human molecular genetics [Hum Mol Genet] 2018 Feb 01; Vol. 27 (3), pp. 407-420. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: IRL Press at Oxford University Press Country of Publication: England NLM ID: 9208958 Publication Model: Print Cited Medium: Internet ISSN: 1460-2083 (Electronic) Linking ISSN: 09646906 NLM ISO Abbreviation: Hum Mol Genet Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
|
Full text is not displayed to guests.
Login for full access.
|
|
| ISSN: | 1460-2083 |
|---|---|
| DOI: | 10.1093/hmg/ddx405 |
