Phosphorylation of MCAD selectively rescues PINK1 deficiencies in behavior and metabolism.

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Title: Phosphorylation of MCAD selectively rescues PINK1 deficiencies in behavior and metabolism.
Authors: Course MM; Department of Neurosurgery, Stanford University, Stanford, CA 94305.; Neurosciences Graduate Program, Stanford University, Stanford, CA 94305., Scott AI; Department of Pathology, Stanford University, Stanford, CA 94305., Schoor C; Institute for Biochemistry and Molecular Biology, University of Bonn, 53115 Bonn, Germany., Hsieh CH; Department of Neurosurgery, Stanford University, Stanford, CA 94305., Papakyrikos AM; Department of Neurosurgery, Stanford University, Stanford, CA 94305.; Developmental Biology Graduate Program, Stanford University, Stanford, CA 94305., Winter D; Institute for Biochemistry and Molecular Biology, University of Bonn, 53115 Bonn, Germany., Cowan TM; Department of Pathology, Stanford University, Stanford, CA 94305., Wang X; Department of Neurosurgery, Stanford University, Stanford, CA 94305.
Source: Molecular biology of the cell [Mol Biol Cell] 2018 May 15; Vol. 29 (10), pp. 1219-1227. Date of Electronic Publication: 2018 Mar 22.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: American Society for Cell Biology Country of Publication: United States NLM ID: 9201390 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1939-4586 (Electronic) Linking ISSN: 10591524 NLM ISO Abbreviation: Mol Biol Cell Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Phosphorylation of MCAD selectively rescues PINK1 deficiencies in behavior and metabolism.
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  Data: <searchLink fieldCode="AU" term="%22Course+MM%22">Course MM</searchLink>; Department of Neurosurgery, Stanford University, Stanford, CA 94305.; Neurosciences Graduate Program, Stanford University, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Scott+AI%22">Scott AI</searchLink>; Department of Pathology, Stanford University, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Schoor+C%22">Schoor C</searchLink>; Institute for Biochemistry and Molecular Biology, University of Bonn, 53115 Bonn, Germany.<br /><searchLink fieldCode="AU" term="%22Hsieh+CH%22">Hsieh CH</searchLink>; Department of Neurosurgery, Stanford University, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Papakyrikos+AM%22">Papakyrikos AM</searchLink>; Department of Neurosurgery, Stanford University, Stanford, CA 94305.; Developmental Biology Graduate Program, Stanford University, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Winter+D%22">Winter D</searchLink>; Institute for Biochemistry and Molecular Biology, University of Bonn, 53115 Bonn, Germany.<br /><searchLink fieldCode="AU" term="%22Cowan+TM%22">Cowan TM</searchLink>; Department of Pathology, Stanford University, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Wang+X%22">Wang X</searchLink>; Department of Neurosurgery, Stanford University, Stanford, CA 94305.
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  Data: <searchLink fieldCode="JN" term="%229201390%22">Molecular biology of the cell</searchLink> [Mol Biol Cell] 2018 May 15; Vol. 29 (10), pp. 1219-1227. <i>Date of Electronic Publication: </i>2018 Mar 22.
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  Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Society+for+Cell+Biology%22">American Society for Cell Biology </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>9201390 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1939-4586 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210591524%22">10591524 </searchLink><i>NLM ISO Abbreviation: </i>Mol Biol Cell <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=29563254
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        Value: 10.1091/mbc.E18-03-0155
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      – TitleFull: Phosphorylation of MCAD selectively rescues PINK1 deficiencies in behavior and metabolism.
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              Text: 2018 May 15
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              Y: 2018
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