Author Correction: Disruption of the beclin 1-BCL2 autophagy regulatory complex promotes longevity in mice.

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Bibliographic Details
Title: Author Correction: Disruption of the beclin 1-BCL2 autophagy regulatory complex promotes longevity in mice.
Authors: Fernández ÁF; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Sebti S; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Wei Y; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA., Zou Z; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA., Shi M; Charles and Jane Pak Center for Mineral Metabolism and Clinical Research, University of Texas Southwestern Medical Center, Dallas, TX, USA., McMillan KL; Charles and Jane Pak Center for Mineral Metabolism and Clinical Research, University of Texas Southwestern Medical Center, Dallas, TX, USA., He C; Department of Cell and Molecular Biology, Feinberg School of Medicine, Northwestern University, Chicago, IL, USA., Ting T; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Liu Y; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA., Chiang WC; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Marciano DK; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Schiattarella GG; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA., Bhagat G; Department of Pathology and Cell Biology, Columbia University Medical Center and New York Presbyterian Hospital, New York, NY, USA., Moe OW; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Charles and Jane Pak Center for Mineral Metabolism and Clinical Research, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Physiology, University of Texas Southwestern Medical Center, Dallas, TX, USA., Hu MC; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA. ming-chang.hu@utsouthwestern.edu.; Charles and Jane Pak Center for Mineral Metabolism and Clinical Research, University of Texas Southwestern Medical Center, Dallas, TX, USA. ming-chang.hu@utsouthwestern.edu., Levine B; Center for Autophagy Research, University of Texas Southwestern Medical Center, Dallas, TX, USA. beth.levine@utsouthwestern.edu.; Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA. beth.levine@utsouthwestern.edu.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA. beth.levine@utsouthwestern.edu.; Department of Microbiology, University of Texas Southwestern Medical Center, Dallas, TX, USA. beth.levine@utsouthwestern.edu.
Source: Nature [Nature] 2018 Sep; Vol. 561 (7723), pp. E30.
Publication Type: Journal Article; Published Erratum
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 0410462 Publication Model: Print Cited Medium: Internet ISSN: 1476-4687 (Electronic) Linking ISSN: 00280836 NLM ISO Abbreviation: Nature Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1476-4687
DOI:10.1038/s41586-018-0270-4