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Prion protein interacts with the metabotropic glutamate receptor 1 and regulates the organization of Ca2+ signaling.

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Title: Prion protein interacts with the metabotropic glutamate receptor 1 and regulates the organization of Ca2+ signaling.
Authors: Matsubara T; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Okayama University Hospital Biobank, Okayama University Hospital, Okayama, Japan., Satoh K; Department of Locomotive Rehabilitation Science, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan., Homma T; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Department of Biochemistry and Molecular Biology, Graduate School of Medical Science, Yamagata University, Japan., Nakagaki T; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan., Yamaguchi N; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan., Atarashi R; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Division of Microbiology, Department of Infectious Diseases, Faculty of Medicine, University of Miyazaki, Japan., Sudo Y; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Cancer Pathophysiology Division, National Cancer Center, Research Institute, Japan., Uezono Y; Cancer Pathophysiology Division, National Cancer Center, Research Institute, Japan., Ishibashi D; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan. Electronic address: dishi@nagasaki-u.ac.jp., Nishida N; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan.
Source: Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2020 Apr 30; Vol. 525 (2), pp. 447-454. Date of Electronic Publication: 2020 Feb 24.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier Country of Publication: United States NLM ID: 0372516 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1090-2104 (Electronic) Linking ISSN: 0006291X NLM ISO Abbreviation: Biochem Biophys Res Commun Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Prion protein interacts with the metabotropic glutamate receptor 1 and regulates the organization of Ca<superscript>2+</superscript> signaling.
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  Data: <searchLink fieldCode="AU" term="%22Matsubara+T%22">Matsubara T</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Okayama University Hospital Biobank, Okayama University Hospital, Okayama, Japan.<br /><searchLink fieldCode="AU" term="%22Satoh+K%22">Satoh K</searchLink>; Department of Locomotive Rehabilitation Science, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan.<br /><searchLink fieldCode="AU" term="%22Homma+T%22">Homma T</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Department of Biochemistry and Molecular Biology, Graduate School of Medical Science, Yamagata University, Japan.<br /><searchLink fieldCode="AU" term="%22Nakagaki+T%22">Nakagaki T</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan.<br /><searchLink fieldCode="AU" term="%22Yamaguchi+N%22">Yamaguchi N</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan.<br /><searchLink fieldCode="AU" term="%22Atarashi+R%22">Atarashi R</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Division of Microbiology, Department of Infectious Diseases, Faculty of Medicine, University of Miyazaki, Japan.<br /><searchLink fieldCode="AU" term="%22Sudo+Y%22">Sudo Y</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan; Cancer Pathophysiology Division, National Cancer Center, Research Institute, Japan.<br /><searchLink fieldCode="AU" term="%22Uezono+Y%22">Uezono Y</searchLink>; Cancer Pathophysiology Division, National Cancer Center, Research Institute, Japan.<br /><searchLink fieldCode="AU" term="%22Ishibashi+D%22">Ishibashi D</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan. Electronic address: dishi@nagasaki-u.ac.jp.<br /><searchLink fieldCode="AU" term="%22Nishida+N%22">Nishida N</searchLink>; Department of Molecular Microbiology and Immunology, Nagasaki University Graduate School of Biomedical Sciences, Japan.
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  Data: <searchLink fieldCode="JN" term="%220372516%22">Biochemical and biophysical research communications</searchLink> [Biochem Biophys Res Commun] 2020 Apr 30; Vol. 525 (2), pp. 447-454. <i>Date of Electronic Publication: </i>2020 Feb 24.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier%22">Elsevier </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>0372516 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1090-2104 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%220006291X%22">0006291X </searchLink><i>NLM ISO Abbreviation: </i>Biochem Biophys Res Commun <i>Subsets: </i>MEDLINE
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