View in EDS

Pirfenidone Is an Agonistic Ligand for PPARα and Improves NASH by Activation of SIRT1/LKB1/pAMPK.

Saved in:
Bibliographic Details
Title: Pirfenidone Is an Agonistic Ligand for PPARα and Improves NASH by Activation of SIRT1/LKB1/pAMPK.
Authors: Sandoval-Rodriguez A; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Monroy-Ramirez HC; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Meza-Rios A; Tecnologico de Monterrey Campus Guadalajara Zapopan México., Garcia-Bañuelos J; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Vera-Cruz J; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Gutiérrez-Cuevas J; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Silva-Gomez J; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Staels B; Institut Pasteur de Lille Universite Lille Inserm, CHU Lille U1011-EGID Lille France., Dominguez-Rosales J; Chronic-Degenerative Diseases Institute Health Sciences University Center University of Guadalajara Guadalajara México., Galicia-Moreno M; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México., Vazquez-Del Mercado M; IIRSME CUCS University of Guadalajara Guadalajara México., Navarro-Partida J; Tecnologico de Monterrey Campus Guadalajara Zapopan México., Santos-Garcia A; Tecnologico de Monterrey Campus Guadalajara Zapopan México., Armendariz-Borunda J; Department of Molecular Biology and Genomics Institute for Molecular Biology in Medicine and Gene Therapy Health Sciences University Center University of Guadalajara Guadalajara México.; Tecnologico de Monterrey Campus Guadalajara Zapopan México.
Source: Hepatology communications [Hepatol Commun] 2020 Jan 16; Vol. 4 (3), pp. 434-449. Date of Electronic Publication: 2020 Jan 16 (Print Publication: 2020).
Publication Type: Journal Article
Journal Info: Publisher: Wolters Kluwer Health, Inc Country of Publication: United States NLM ID: 101695860 Publication Model: eCollection Cited Medium: Internet ISSN: 2471-254X (Electronic) Linking ISSN: 2471254X NLM ISO Abbreviation: Hepatol Commun Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2471-254X
DOI:10.1002/hep4.1474