Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity.

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Bibliographic Details
Title: Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity.
Authors: Jestin M; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Kapnick SM; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Tarasenko TN; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Burke CT; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Zerfas PM; Office of Research Services, National Institutes of Health, Bethesda, MD, 20892, USA., Diaz F; University of Miami, Department of Neurology, Miller School of Medicine, Miami, FL, 33136, USA., Vernon H; Kennedy Krieger Institute, Johns Hopkins Medical Center, Baltimore, MD, 21205, USA., Singh LN; Center for Mitochondrial and Epigenomic Medicine, Children's Hospital of Philadelphia, Philadelphia, PA, 19104, USA., Sokol RJ; Section of Pediatric Gastroenterology, Hepatology and Nutrition, University of Colorado School of Medicine and Children's Hospital Colorado, Aurora, CO 80045, USA., McGuire PJ; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA. Electronic address: peter.mcguire@nih.gov.
Source: Molecular metabolism [Mol Metab] 2020 Jul; Vol. 37, pp. 100981. Date of Electronic Publication: 2020 Mar 26.
Publication Type: Journal Article; Research Support, N.I.H., Intramural
Journal Info: Publisher: Elsevier GmbH Country of Publication: Germany NLM ID: 101605730 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2212-8778 (Electronic) Linking ISSN: 22128778 NLM ISO Abbreviation: Mol Metab Subsets: MEDLINE
Database: MEDLINE Ultimate
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