p53 destabilizing protein skews asymmetric division and enhances NOTCH activation to direct self-renewal of TICs.
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| Title: | p53 destabilizing protein skews asymmetric division and enhances NOTCH activation to direct self-renewal of TICs. |
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| Authors: | Choi HY; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Siddique HR; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA.; Section of Genetics, Department of Zoology, Aligarh Muslim University, Aligarh, 202002, India., Zheng M; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Kou Y; Department of Chemistry and Biological Sciences, University of Southern California, Los Angeles, CA, 90089, USA., Yeh DW; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Machida T; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Chen CL; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Uthaya Kumar DB; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA.; Department of Genetics and Genomics, and The Jackson Laboratory for Genomic Medicine, University of Connecticut Health, Farmington, 06032, CT, USA., Punj V; Department of Medicine, University of Southern California, Los Angeles, CA, 90033, USA., Winer P; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Pita A; Department of Surgery, University of Southern California, Los Angeles, CA, 90033, USA., Sher L; Department of Surgery, University of Southern California, Los Angeles, CA, 90033, USA., Tahara SM; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Ray RB; Saint Louis University, St. Louis, MO, 63103, USA., Liang C; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA., Chen L; Department of Chemistry and Biological Sciences, University of Southern California, Los Angeles, CA, 90089, USA., Tsukamoto H; Department of Pathology, Keck School of Medicine, University of Southern California, Los Angeles, CA, 90033, USA.; Southern California Research Center for ALPD and Cirrhosis, Los Angeles, CA, 90033, USA.; VA Greater Los Angeles Healthcare System, Los Angeles, 90073, CA, USA., Machida K; Department of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, 90033, USA. keigo.machida@med.usc.edu.; Southern California Research Center for ALPD and Cirrhosis, Los Angeles, CA, 90033, USA. keigo.machida@med.usc.edu. |
| Source: | Nature communications [Nat Commun] 2020 Jun 17; Vol. 11 (1), pp. 3084. Date of Electronic Publication: 2020 Jun 17. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 2041-1723 |
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| DOI: | 10.1038/s41467-020-16616-8 |
