Age-related loss of neural stem cell O-GlcNAc promotes a glial fate switch through STAT3 activation.

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Bibliographic Details
Title: Age-related loss of neural stem cell O-GlcNAc promotes a glial fate switch through STAT3 activation.
Authors: White CW 3rd; Department of Anatomy, University of California, San Francisco, CA 94143.; Developmental and Stem Cell Biology Graduate Program, University of California, San Francisco, CA 94143., Fan X; Department of Anatomy, University of California, San Francisco, CA 94143., Maynard JC; Mass Spectrometry Facility, Department of Pharmaceutical Chemistry, University of California, San Francisco, CA 94158., Wheatley EG; Department of Anatomy, University of California, San Francisco, CA 94143.; Developmental and Stem Cell Biology Graduate Program, University of California, San Francisco, CA 94143., Bieri G; Department of Anatomy, University of California, San Francisco, CA 94143., Couthouis J; Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305., Burlingame AL; Mass Spectrometry Facility, Department of Pharmaceutical Chemistry, University of California, San Francisco, CA 94158., Villeda SA; Department of Anatomy, University of California, San Francisco, CA 94143; Saul.Villeda@ucsf.edu.; Developmental and Stem Cell Biology Graduate Program, University of California, San Francisco, CA 94143.; Department of Physical Therapy and Rehabilitation Science, University of California, San Francisco, CA 94143.; The Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, CA 94143.
Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 Sep 08; Vol. 117 (36), pp. 22214-22224. Date of Electronic Publication: 2020 Aug 26.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1091-6490 (Electronic) Linking ISSN: 00278424 NLM ISO Abbreviation: Proc Natl Acad Sci U S A Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1091-6490
DOI:10.1073/pnas.2007439117