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YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction.

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Title: YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction.
Authors: Mia MM; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore., Cibi DM; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore., Abdul Ghani SAB; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore., Song W; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore., Tee N; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore., Ghosh S; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore., Mao J; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America., Olson EN; Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, Texas, United States of America., Singh MK; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore.
Source: PLoS biology [PLoS Biol] 2020 Dec 02; Vol. 18 (12), pp. e3000941. Date of Electronic Publication: 2020 Dec 02 (Print Publication: 2020).
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101183755 Publication Model: eCollection Cited Medium: Internet ISSN: 1545-7885 (Electronic) Linking ISSN: 15449173 NLM ISO Abbreviation: PLoS Biol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction.
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  Data: <searchLink fieldCode="AU" term="%22Mia+MM%22">Mia MM</searchLink>; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.<br /><searchLink fieldCode="AU" term="%22Cibi+DM%22">Cibi DM</searchLink>; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.<br /><searchLink fieldCode="AU" term="%22Abdul+Ghani+SAB%22">Abdul Ghani SAB</searchLink>; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.<br /><searchLink fieldCode="AU" term="%22Song+W%22">Song W</searchLink>; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Tee+N%22">Tee N</searchLink>; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Ghosh+S%22">Ghosh S</searchLink>; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.<br /><searchLink fieldCode="AU" term="%22Mao+J%22">Mao J</searchLink>; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.<br /><searchLink fieldCode="AU" term="%22Olson+EN%22">Olson EN</searchLink>; Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, Texas, United States of America.<br /><searchLink fieldCode="AU" term="%22Singh+MK%22">Singh MK</searchLink>; Cardiovascular and Metabolic Disorders Program, Duke-NUS Medical School Singapore. Singapore.; National Heart Research Institute Singapore, National Heart Centre Singapore, Singapore.
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  Data: <searchLink fieldCode="JN" term="%22101183755%22">PLoS biology</searchLink> [PLoS Biol] 2020 Dec 02; Vol. 18 (12), pp. e3000941. <i>Date of Electronic Publication: </i>2020 Dec 02 (<i>Print Publication: </i>2020).
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        Value: 10.1371/journal.pbio.3000941
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              Text: 2020 Dec 02
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