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Immunosuppressive Myeloid Cells Induce Nitric Oxide-Dependent DNA Damage and p53 Pathway Activation in CD8+ T Cells.

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Title: Immunosuppressive Myeloid Cells Induce Nitric Oxide-Dependent DNA Damage and p53 Pathway Activation in CD8+ T Cells.
Authors: Cartwright ANR; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts., Suo S; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts., Badrinath S; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts., Kumar S; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts., Melms J; Columbia Center for Translational Immunology, New York, New York.; Columbia University Medical Center, Division of Hematology and Oncology, New York, New York., Luoma A; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts., Bagati A; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts., Saadatpour A; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts., Izar B; Columbia Center for Translational Immunology, New York, New York.; Columbia University Medical Center, Division of Hematology and Oncology, New York, New York., Yuan GC; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts., Wucherpfennig KW; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts. kai_wucherpfennig@dfci.harvard.edu.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.; Department of Neurology, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.
Source: Cancer immunology research [Cancer Immunol Res] 2021 Apr; Vol. 9 (4), pp. 470-485. Date of Electronic Publication: 2021 Jan 29.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Association for Cancer Research Country of Publication: United States NLM ID: 101614637 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2326-6074 (Electronic) Linking ISSN: 23266066 NLM ISO Abbreviation: Cancer Immunol Res Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Immunosuppressive Myeloid Cells Induce Nitric Oxide-Dependent DNA Damage and p53 Pathway Activation in CD8<superscript>+</superscript> T Cells.
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  Data: <searchLink fieldCode="AU" term="%22Cartwright+ANR%22">Cartwright ANR</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Suo+S%22">Suo S</searchLink>; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Badrinath+S%22">Badrinath S</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Kumar+S%22">Kumar S</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Melms+J%22">Melms J</searchLink>; Columbia Center for Translational Immunology, New York, New York.; Columbia University Medical Center, Division of Hematology and Oncology, New York, New York.<br /><searchLink fieldCode="AU" term="%22Luoma+A%22">Luoma A</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Bagati+A%22">Bagati A</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Saadatpour+A%22">Saadatpour A</searchLink>; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Izar+B%22">Izar B</searchLink>; Columbia Center for Translational Immunology, New York, New York.; Columbia University Medical Center, Division of Hematology and Oncology, New York, New York.<br /><searchLink fieldCode="AU" term="%22Yuan+GC%22">Yuan GC</searchLink>; Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.<br /><searchLink fieldCode="AU" term="%22Wucherpfennig+KW%22">Wucherpfennig KW</searchLink>; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, Massachusetts. kai_wucherpfennig@dfci.harvard.edu.; Department of Immunology, Harvard Medical School, Boston, Massachusetts.; Department of Neurology, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.
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  Data: <searchLink fieldCode="JN" term="%22101614637%22">Cancer immunology research</searchLink> [Cancer Immunol Res] 2021 Apr; Vol. 9 (4), pp. 470-485. <i>Date of Electronic Publication: </i>2021 Jan 29.
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