The pro-inflammatory microRNA miR-155 influences fibrillar β-Amyloid1-42 catabolism by microglia.

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Title: The pro-inflammatory microRNA miR-155 influences fibrillar β-Amyloid1-42 catabolism by microglia.
Authors: Aloi MS; Department of Pathology, School of Medicine, University of Washington, Seattle, Washington, USA.; Department of Neurology, Baylor College of Medicine, Houston, Texas, USA., Prater KE; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA., Sopher B; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA., Davidson S; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA., Jayadev S; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA., Garden GA; Department of Pathology, School of Medicine, University of Washington, Seattle, Washington, USA.; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.
Source: Glia [Glia] 2021 Jul; Vol. 69 (7), pp. 1736-1748. Date of Electronic Publication: 2021 Mar 10.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Wiley-Liss Country of Publication: United States NLM ID: 8806785 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1098-1136 (Electronic) Linking ISSN: 08941491 NLM ISO Abbreviation: Glia Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: The pro-inflammatory microRNA miR-155 influences fibrillar β-Amyloid<subscript>1</subscript><subscript>-42</subscript> catabolism by microglia.
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  Data: <searchLink fieldCode="AU" term="%22Aloi+MS%22">Aloi MS</searchLink>; Department of Pathology, School of Medicine, University of Washington, Seattle, Washington, USA.; Department of Neurology, Baylor College of Medicine, Houston, Texas, USA.<br /><searchLink fieldCode="AU" term="%22Prater+KE%22">Prater KE</searchLink>; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.<br /><searchLink fieldCode="AU" term="%22Sopher+B%22">Sopher B</searchLink>; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.<br /><searchLink fieldCode="AU" term="%22Davidson+S%22">Davidson S</searchLink>; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.<br /><searchLink fieldCode="AU" term="%22Jayadev+S%22">Jayadev S</searchLink>; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.<br /><searchLink fieldCode="AU" term="%22Garden+GA%22">Garden GA</searchLink>; Department of Pathology, School of Medicine, University of Washington, Seattle, Washington, USA.; Department of Neurology, School of Medicine, University of Washington, Seattle, Washington, USA.
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  Data: <searchLink fieldCode="JN" term="%228806785%22">Glia</searchLink> [Glia] 2021 Jul; Vol. 69 (7), pp. 1736-1748. <i>Date of Electronic Publication: </i>2021 Mar 10.
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  Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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        Value: 10.1002/glia.23988
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      – TitleFull: The pro-inflammatory microRNA miR-155 influences fibrillar β-Amyloid1-42 catabolism by microglia.
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              Text: 2021 Jul
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