Therapeutically expanded human regulatory T-cells are super-suppressive due to HIF1A induced expression of CD73.
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| Title: | Therapeutically expanded human regulatory T-cells are super-suppressive due to HIF1A induced expression of CD73. |
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| Authors: | Jarvis LB; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Rainbow DB; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.; JDRF/Wellcome Diabetes and Inflammation Laboratory, Wellcome Centre for Human Genetics, Nuffield Department of Medicine, NIHR Oxford Biomedical Research Centre, University of Oxford, Oxford, UK., Coppard V; Department of Medicine, University of Cambridge, Cambridge, UK., Howlett SK; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Georgieva Z; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Davies JL; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Mullay HK; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Hester J; Department of Nuffield Department of Surgical Sciences, University of Oxford, Oxford, UK., Ashmore T; Department of Biochemistry and Cambridge Systems Biology Centre, University of Cambridge, Cambridge, UK., Van Den Bosch A; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Grist JT; Department of Radiology, University of Cambridge, Cambridge, UK., Coles AJ; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Mousa HS; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Pluchino S; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Mahbubani KT; Department of Surgery, University of Cambridge, Cambridge, UK., Griffin JL; Department of Biochemistry and Cambridge Systems Biology Centre, University of Cambridge, Cambridge, UK.; Imperial College London Dementia Research Institute & Section of Biomolecular Medicine, Department of Metabolism, Digestion and Reproduction, Imperial College London, London, UK., Saeb-Parsy K; Department of Surgery, University of Cambridge, Cambridge, UK., Issa F; Department of Nuffield Department of Surgical Sciences, University of Oxford, Oxford, UK., Peruzzotti-Jametti L; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Wicker LS; JDRF/Wellcome Diabetes and Inflammation Laboratory, Wellcome Centre for Human Genetics, Nuffield Department of Medicine, NIHR Oxford Biomedical Research Centre, University of Oxford, Oxford, UK., Jones JL; Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK. jls53@medschl.cam.ac.uk. |
| Source: | Communications biology [Commun Biol] 2021 Oct 14; Vol. 4 (1), pp. 1186. Date of Electronic Publication: 2021 Oct 14. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Publishing Group UK Country of Publication: England NLM ID: 101719179 Publication Model: Electronic Cited Medium: Internet ISSN: 2399-3642 (Electronic) Linking ISSN: 23993642 NLM ISO Abbreviation: Commun Biol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 2399-3642 |
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| DOI: | 10.1038/s42003-021-02721-x |
