S100A9 is a functional effector of infarct wall thinning after myocardial infarction.

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Bibliographic Details
Title: S100A9 is a functional effector of infarct wall thinning after myocardial infarction.
Authors: Chalise U; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Research Service, Nebraska-Western Iowa Health Care System, Omaha, Nebraska., Becirovic-Agic M; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Research Service, Nebraska-Western Iowa Health Care System, Omaha, Nebraska., Daseke MJ 2nd; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Research Service, Nebraska-Western Iowa Health Care System, Omaha, Nebraska.; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi., Konfrst SR; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Research Service, Nebraska-Western Iowa Health Care System, Omaha, Nebraska., Rodriguez-Paar JR; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska., Feng D; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Division of Pediatric Critical Care, Department of Pediatrics, University of Nebraska Medical Center, Omaha, Nebraska., Salomon JD; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Division of Pediatric Critical Care, Department of Pediatrics, University of Nebraska Medical Center, Omaha, Nebraska., Anderson DR; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Division of Cardiovascular Medicine, Department of Internal Medicine, University of Nebraska Medical Center, Omaha, Nebraska., Cook LM; Department of Pathology and Microbiology, University of Nebraska Medical Center, Omaha, Nebraska., Lindsey ML; Department of Cellular and Integrative Physiology, Center for Heart and Vascular Research, University of Nebraska Medical Center, Omaha, Nebraska.; Research Service, Nebraska-Western Iowa Health Care System, Omaha, Nebraska.
Source: American journal of physiology. Heart and circulatory physiology [Am J Physiol Heart Circ Physiol] 2022 Feb 01; Vol. 322 (2), pp. H145-H155. Date of Electronic Publication: 2021 Dec 10.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901228 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1539 (Electronic) Linking ISSN: 03636135 NLM ISO Abbreviation: Am J Physiol Heart Circ Physiol Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1522-1539
DOI:10.1152/ajpheart.00475.2021