ATF-4 and hydrogen sulfide signalling mediate longevity in response to inhibition of translation or mTORC1.
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| Title: | ATF-4 and hydrogen sulfide signalling mediate longevity in response to inhibition of translation or mTORC1. |
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| Authors: | Statzer C; Eidgenössische Technische Hochschule Zürich, Department of Health Sciences and Technology, Institute of Translational Medicine, Schwerzenbach, Switzerland., Meng J; Department of Genetics, Harvard Medical School, Boston, MA, USA.; Joslin Diabetes Center, Research Division, Boston, MA, USA.; Harvard Stem Cell Institute, Cambridge, MA, USA., Venz R; Eidgenössische Technische Hochschule Zürich, Department of Health Sciences and Technology, Institute of Translational Medicine, Schwerzenbach, Switzerland., Bland M; Department of Genetics, Harvard Medical School, Boston, MA, USA.; Joslin Diabetes Center, Research Division, Boston, MA, USA.; Harvard Stem Cell Institute, Cambridge, MA, USA., Robida-Stubbs S; Department of Genetics, Harvard Medical School, Boston, MA, USA.; Joslin Diabetes Center, Research Division, Boston, MA, USA.; Harvard Stem Cell Institute, Cambridge, MA, USA., Patel K; Department of Genetics, Harvard Medical School, Boston, MA, USA.; Joslin Diabetes Center, Research Division, Boston, MA, USA.; Harvard Stem Cell Institute, Cambridge, MA, USA., Petrovic D; Leibniz-Institut für Analytische Wissenschaften-ISAS-e.V., Dortmund, Germany., Emsley R; Department of Vascular Surgery, Centre Hospitalier Universitaire Vaudois and University of Lausanne, Lausanne, Switzerland., Liu P; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA, USA., Morantte I; Department of Genetics and Complex Diseases, Harvard School of Public Health, 665 Huntington Avenue, Boston, MA, USA., Haynes C; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA, USA., Mair WB; Department of Genetics and Complex Diseases, Harvard School of Public Health, 665 Huntington Avenue, Boston, MA, USA., Longchamp A; Department of Vascular Surgery, Centre Hospitalier Universitaire Vaudois and University of Lausanne, Lausanne, Switzerland., Filipovic MR; Leibniz-Institut für Analytische Wissenschaften-ISAS-e.V., Dortmund, Germany., Blackwell TK; Department of Genetics, Harvard Medical School, Boston, MA, USA. keith.blackwell@joslin.harvard.edu.; Joslin Diabetes Center, Research Division, Boston, MA, USA. keith.blackwell@joslin.harvard.edu.; Harvard Stem Cell Institute, Cambridge, MA, USA. keith.blackwell@joslin.harvard.edu., Ewald CY; Eidgenössische Technische Hochschule Zürich, Department of Health Sciences and Technology, Institute of Translational Medicine, Schwerzenbach, Switzerland. collin-ewald@ethz.ch. |
| Source: | Nature communications [Nat Commun] 2022 Feb 18; Vol. 13 (1), pp. 967. Date of Electronic Publication: 2022 Feb 18. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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