Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics.

Saved in:
Bibliographic Details
Title: Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics.
Authors: Iida T; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan.; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Mizutani Y; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan.; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Esaki N; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Ponik SM; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, WI, USA., Burkel BM; Department of Cell and Regenerative Biology, University of Wisconsin-Madison, Madison, WI, USA., Weng L; Department of Oncology, Xiangya Cancer Center, Xiangya Hospital, Central South University, Changsha, China., Kuwata K; Institute of Transformative Bio-Molecules, Nagoya University, Nagoya, Japan., Masamune A; Division of Gastroenterology, Tohoku University Graduate School of Medicine, Sendai, Japan., Ishihara S; Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan., Haga H; Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan., Kataoka K; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan.; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Mii S; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Shiraki Y; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Ishikawa T; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Ohno E; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan., Kawashima H; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.; Department of Endoscopy, Nagoya University Hospital, Nagoya, Japan., Hirooka Y; Department of Gastroenterology and Hepatology, Fujita Health University, Toyoake, Aichi, Japan., Fujishiro M; Department of Gastroenterology and Hepatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.; Department of Gastroenterology, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Takahashi M; International Center for Cell and Gene Therapy, Fujita Health University, Toyoake, Japan., Enomoto A; Department of Pathology, Nagoya University Graduate School of Medicine, Nagoya, Japan. enomoto@iar.nagoya-u.ac.jp.
Source: Oncogene [Oncogene] 2022 May; Vol. 41 (19), pp. 2764-2777. Date of Electronic Publication: 2022 Apr 13.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8711562 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1476-5594 (Electronic) Linking ISSN: 09509232 NLM ISO Abbreviation: Oncogene Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1476-5594
DOI:10.1038/s41388-022-02288-9