P2RY2-AKT activation is a therapeutically actionable consequence of XPO1 inhibition in acute myeloid leukemia.

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Title: P2RY2-AKT activation is a therapeutically actionable consequence of XPO1 inhibition in acute myeloid leukemia.
Authors: Lin KH; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Rutter JC; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Xie A; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Killarney ST; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Vaganay C; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Benaksas C; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Ling F; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Sodaro G; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Meslin PA; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Bassil CF; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Fenouille N; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Hoj J; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Washart R; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Ang HX; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Cerda-Smith C; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Chaintreuil P; Université Côte d'Azur, Nice, France., Jacquel A; Université Côte d'Azur, Nice, France., Auberger P; Université Côte d'Azur, Nice, France., Forget A; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Itzykson R; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Lu M; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA., Lin J; Department of Biostatistics and Bioinformatics, Duke University, Durham, NC, USA., Pierobon M; Center for Applied Proteomics and Molecular Medicine, School of Systems Biology, George Mason University, Manassas, VA, USA., Sheng Z; Department of Biostatistics and Bioinformatics, Duke University, Durham, NC, USA., Li X; Department of Biomedical Engineering, Duke University, Durham, NC, USA., Chilkoti A; Department of Biomedical Engineering, Duke University, Durham, NC, USA., Owzar K; Department of Biostatistics and Bioinformatics, Duke University, Durham, NC, USA., Rizzieri DA; Department of Medicine, Duke University Medical Center, Durham, NC, USA., Pardee TS; Department of Internal Medicine, Section on Hematology and Oncology, Wake Forest Baptist Health, Winston-Salem, NC, USA., Benajiba L; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France., Petricoin E; Center for Applied Proteomics and Molecular Medicine, School of Systems Biology, George Mason University, Manassas, VA, USA., Puissant A; Université de Paris, Génomes, Biologie Cellulaire et Thérapeutique U944, INSERM, CNRS, Paris, France. alexandre.puissant@inserm.fr., Wood KC; Department of Pharmacology and Cancer Biology, Duke University, Durham, NC, USA. kris.wood@duke.edu.
Source: Nature cancer [Nat Cancer] 2022 Jul; Vol. 3 (7), pp. 837-851. Date of Electronic Publication: 2022 Jun 06.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 101761119 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2662-1347 (Electronic) Linking ISSN: 26621347 NLM ISO Abbreviation: Nat Cancer Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2662-1347
DOI:10.1038/s43018-022-00394-x