Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma.
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| Title: | Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma. |
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| Authors: | Thadathil N; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Selvarani R; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Mohammed S; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Nicklas EH; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Tran AL; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Kamal M; Department of Pathology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Luo W; Department of Pathology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Brown JL; Aging & Metabolism Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.; Oklahoma City VA Medical Center, Oklahoma City, Oklahoma, USA., Lawrence MM; Aging & Metabolism Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.; Department of Kinesiology and Outdoor Recreation, Southern Utah University, Cedar City, Utah, USA., Borowik AK; Aging & Metabolism Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA., Miller BF; Aging & Metabolism Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.; Oklahoma City VA Medical Center, Oklahoma City, Oklahoma, USA.; Center for Geroscience and Healthy Brain Aging, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Van Remmen H; Aging & Metabolism Program, Oklahoma Medical Research Foundation, Oklahoma City, Oklahoma, USA.; Oklahoma City VA Medical Center, Oklahoma City, Oklahoma, USA.; Center for Geroscience and Healthy Brain Aging, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Richardson A; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.; Oklahoma City VA Medical Center, Oklahoma City, Oklahoma, USA.; Center for Geroscience and Healthy Brain Aging, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA., Deepa SS; Department of Biochemistry & Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA.; Center for Geroscience and Healthy Brain Aging, University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma, USA. |
| Source: | Aging cell [Aging Cell] 2022 Aug; Vol. 21 (8), pp. e13676. Date of Electronic Publication: 2022 Jul 23. |
| Publication Type: | Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, N.I.H., Extramural |
| Journal Info: | Publisher: Wiley-Blackwell Country of Publication: England NLM ID: 101130839 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1474-9726 (Electronic) Linking ISSN: 14749718 NLM ISO Abbreviation: Aging Cell Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 1474-9726 |
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| DOI: | 10.1111/acel.13676 |
