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Age-related Huntington's disease progression modeled in directly reprogrammed patient-derived striatal neurons highlights impaired autophagy.

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Title: Age-related Huntington's disease progression modeled in directly reprogrammed patient-derived striatal neurons highlights impaired autophagy.
Authors: Oh YM; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Lee SW; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Kim WK; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Chen S; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Church VA; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Cates K; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA., Li T; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA., Zhang B; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA., Dolle RE; Department of Biochemistry, Washington University School of Medicine, St. Louis, MO, USA., Dahiya S; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO, USA., Pak SC; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA., Silverman GA; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA., Perlmutter DH; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA., Yoo AS; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA. yooa@wustl.edu.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA. yooa@wustl.edu.
Source: Nature neuroscience [Nat Neurosci] 2022 Nov; Vol. 25 (11), pp. 1420-1433. Date of Electronic Publication: 2022 Oct 27.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
Journal Info: Publisher: Nature Publishing Group Country of Publication: United States NLM ID: 9809671 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1546-1726 (Electronic) Linking ISSN: 10976256 NLM ISO Abbreviation: Nat Neurosci Subsets: MEDLINE
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  Data: Age-related Huntington's disease progression modeled in directly reprogrammed patient-derived striatal neurons highlights impaired autophagy.
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  Data: <searchLink fieldCode="AU" term="%22Oh+YM%22">Oh YM</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Lee+SW%22">Lee SW</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Kim+WK%22">Kim WK</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Chen+S%22">Chen S</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Church+VA%22">Church VA</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Cates+K%22">Cates K</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Li+T%22">Li T</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Zhang+B%22">Zhang B</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Dolle+RE%22">Dolle RE</searchLink>; Department of Biochemistry, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Dahiya+S%22">Dahiya S</searchLink>; Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Pak+SC%22">Pak SC</searchLink>; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Silverman+GA%22">Silverman GA</searchLink>; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Perlmutter+DH%22">Perlmutter DH</searchLink>; Department of Pediatrics, Washington University School of Medicine, St. Louis, MO, USA.<br /><searchLink fieldCode="AU" term="%22Yoo+AS%22">Yoo AS</searchLink>; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, USA. yooa@wustl.edu.; Center of Regenerative Medicine, Washington University School of Medicine, St. Louis, MO, USA. yooa@wustl.edu.
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  Data: <searchLink fieldCode="JN" term="%229809671%22">Nature neuroscience</searchLink> [Nat Neurosci] 2022 Nov; Vol. 25 (11), pp. 1420-1433. <i>Date of Electronic Publication: </i>2022 Oct 27.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Publishing+Group%22">Nature Publishing Group </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>9809671 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1546-1726 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210976256%22">10976256 </searchLink><i>NLM ISO Abbreviation: </i>Nat Neurosci <i>Subsets: </i>MEDLINE
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