E2F1-initiated transcription of PRSS22 promotes breast cancer metastasis by cleaving ANXA1 and activating FPR2/ERK signaling pathway.

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Title: E2F1-initiated transcription of PRSS22 promotes breast cancer metastasis by cleaving ANXA1 and activating FPR2/ERK signaling pathway.
Authors: Song L; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Department of Pathology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong, 250021, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Li H; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Ma RR; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Liu S; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Zhang GH; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Guo XY; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Zhao RN; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China., Wu XJ; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China. xiaojuanwu0709@126.com.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China. xiaojuanwu0709@126.com., Zhang K; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China. zk0827019@sina.com.; Department of Breast Surgery, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China. zk0827019@sina.com., Gao P; Department of Pathology, Qilu Hospital of Shandong University, Jinan, Shandong, 250012, China. gaopeng@sdu.edu.cn.; Key Laboratory for Experimental Teratology of the Ministry of Education and Department of Pathology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Jinan, Shandong, 250012, China. gaopeng@sdu.edu.cn.
Source: Cell death & disease [Cell Death Dis] 2022 Nov 21; Vol. 13 (11), pp. 982. Date of Electronic Publication: 2022 Nov 21.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:2041-4889
DOI:10.1038/s41419-022-05414-3