p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol.

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Title: p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol.
Authors: Carlsson MJ; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Vollmer AS; Rudolf Buchheim Institute of Pharmacology, Justus Liebig University Giessen, 35392, Giessen, Germany.; Department of Dermatology, University Medical Center, 69120, Heidelberg, Germany., Demuth P; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Heylmann D; Rudolf Buchheim Institute of Pharmacology, Justus Liebig University Giessen, 35392, Giessen, Germany., Reich D; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany., Quarz C; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Rasenberger B; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany., Nikolova T; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany., Hofmann TG; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany., Christmann M; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany., Fuhlbrueck JA; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Stegmüller S; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Richling E; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Cartus AT; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany., Fahrer J; Division of Food Chemistry and Toxicology, Department of Chemistry, Technical University of Kaiserslautern, 67663, Kaiserslautern, Germany. fahrer@chemie.uni-kl.de.; Rudolf Buchheim Institute of Pharmacology, Justus Liebig University Giessen, 35392, Giessen, Germany. fahrer@chemie.uni-kl.de.; Institute of Toxicology, University Medical Center, Johannes Gutenberg University Mainz, 55131, Mainz, Germany. fahrer@chemie.uni-kl.de.
Source: Cell death & disease [Cell Death Dis] 2022 Nov 29; Vol. 13 (11), pp. 1009. Date of Electronic Publication: 2022 Nov 29.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:2041-4889
DOI:10.1038/s41419-022-05446-9