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Neuronal deletion of MnSOD in mice leads to demyelination, inflammation and progressive paralysis that mimics phenotypes associated with progressive multiple sclerosis.

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Title: Neuronal deletion of MnSOD in mice leads to demyelination, inflammation and progressive paralysis that mimics phenotypes associated with progressive multiple sclerosis.
Authors: Bhaskaran S; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Kumar G; Arthritis & Clinical Immunology, Oklahoma Medical Research Foundation, OK, USA., Thadathil N; Department of Biochemistry and Molecular Biology, University of Oklahoma Health Sciences Center, OK, USA., Piekarz KM; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Mohammed S; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, OK, USA., Lopez SD; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Qaisar R; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Walton D; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Brown JL; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA; Oklahoma City VA Medical Center, Oklahoma City, OK, USA., Murphy A; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Smith N; Advanced Magnetic Resonance Center, Oklahoma Medical Research Foundation, OK, USA., Saunders D; Advanced Magnetic Resonance Center, Oklahoma Medical Research Foundation, OK, USA., Beckstead MJ; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA; Oklahoma City VA Medical Center, Oklahoma City, OK, USA., Plafker S; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Lewis TL Jr; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA., Towner R; Advanced Magnetic Resonance Center, Oklahoma Medical Research Foundation, OK, USA., Deepa SS; Department of Biochemistry and Molecular Biology, University of Oklahoma Health Sciences Center, OK, USA; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, OK, USA., Richardson A; Department of Biochemistry and Molecular Biology, University of Oklahoma Health Sciences Center, OK, USA; Stephenson Cancer Center, University of Oklahoma Health Sciences Center, Oklahoma City, OK, USA; Oklahoma City VA Medical Center, Oklahoma City, OK, USA., Axtell RC; Arthritis & Clinical Immunology, Oklahoma Medical Research Foundation, OK, USA. Electronic address: bob-axtell@omrf.org., Van Remmen H; Aging & Metabolism Research Program, Oklahoma Medical Research Foundation, OK, USA; Oklahoma City VA Medical Center, Oklahoma City, OK, USA. Electronic address: Holly-VanRemmen@omrf.org.
Source: Redox biology [Redox Biol] 2023 Feb; Vol. 59, pp. 102550. Date of Electronic Publication: 2022 Nov 26.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: Elsevier, B.V Country of Publication: Netherlands NLM ID: 101605639 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2213-2317 (Electronic) Linking ISSN: 22132317 NLM ISO Abbreviation: Redox Biol Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2213-2317
DOI:10.1016/j.redox.2022.102550