Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss.
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| Title: | Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss. |
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| Authors: | Dougherty EJ; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Chen LY; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Awad KS; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Ferreyra GA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Demirkale CY; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Keshavarz A; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Gairhe S; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Johnston KA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Hicks ME; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Sandler AB; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Curran CS; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Krack JM; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Ding Y; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Suffredini AF; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Solomon MA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Elinoff JM; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Danner RL; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States. |
| Source: | American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2023 Jun 01; Vol. 324 (6), pp. L783-L798. Date of Electronic Publication: 2023 Apr 11. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Intramural; Research Support, N.I.H., Extramural |
| Journal Info: | Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901229 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1504 (Electronic) Linking ISSN: 10400605 NLM ISO Abbreviation: Am J Physiol Lung Cell Mol Physiol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Links: – Type: pdflink Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 37039367 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Dougherty+EJ%22">Dougherty EJ</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Chen+LY%22">Chen LY</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Awad+KS%22">Awad KS</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Ferreyra+GA%22">Ferreyra GA</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Demirkale+CY%22">Demirkale CY</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Keshavarz+A%22">Keshavarz A</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Gairhe+S%22">Gairhe S</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Johnston+KA%22">Johnston KA</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Hicks+ME%22">Hicks ME</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Sandler+AB%22">Sandler AB</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Curran+CS%22">Curran CS</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Krack+JM%22">Krack JM</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Ding+Y%22">Ding Y</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Suffredini+AF%22">Suffredini AF</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Solomon+MA%22">Solomon MA</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Elinoff+JM%22">Elinoff JM</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States.<br /><searchLink fieldCode="AU" term="%22Danner+RL%22">Danner RL</searchLink>; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22100901229%22">American journal of physiology. Lung cellular and molecular physiology</searchLink> [Am J Physiol Lung Cell Mol Physiol] 2023 Jun 01; Vol. 324 (6), pp. L783-L798. <i>Date of Electronic Publication: </i>2023 Apr 11. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Intramural; Research Support, N.I.H., Extramural – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901229 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1504 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210400605%22">10400605 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Lung Cell Mol Physiol <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=37039367 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1152/ajplung.00171.2022 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: L783 Titles: – TitleFull: Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Dougherty EJ – PersonEntity: Name: NameFull: Chen LY – PersonEntity: Name: NameFull: Awad KS – PersonEntity: Name: NameFull: Ferreyra GA – PersonEntity: Name: NameFull: Demirkale CY – PersonEntity: Name: NameFull: Keshavarz A – PersonEntity: Name: NameFull: Gairhe S – PersonEntity: Name: NameFull: Johnston KA – PersonEntity: Name: NameFull: Hicks ME – PersonEntity: Name: NameFull: Sandler AB – PersonEntity: Name: NameFull: Curran CS – PersonEntity: Name: NameFull: Krack JM – PersonEntity: Name: NameFull: Ding Y – PersonEntity: Name: NameFull: Suffredini AF – PersonEntity: Name: NameFull: Solomon MA – PersonEntity: Name: NameFull: Elinoff JM – PersonEntity: Name: NameFull: Danner RL IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 06 Text: 2023 Jun 01 Type: published Y: 2023 Identifiers: – Type: issn-electronic Value: 1522-1504 Numbering: – Type: volume Value: 324 – Type: issue Value: 6 Titles: – TitleFull: American journal of physiology. Lung cellular and molecular physiology Type: main |
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