IFN-γ and androgens disrupt mitochondrial function in murine myocytes.

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Bibliographic Details
Title: IFN-γ and androgens disrupt mitochondrial function in murine myocytes.
Authors: Fenimore JM; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Springer DA; Murine Phenotyping Core, National Heart, Lung, and Blood Institute, Bethesda, MD, USA., Romero ME; CVPath Institute, Gaithersburg, MD, USA., Edmondson EF; Pathology and Histology Lab, National Cancer Institute, Frederick, MD, USA., McVicar DW; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Yanpallewar S; Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Sanford M; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Spindel T; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Engle E; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Meyer TJ; CCR Collaborative Bioinformatics Resource (CCBR), Center for Cancer Research, National Cancer Institute, Bethesda, MD, USA., Valencia JC; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA., Young HA; Cancer Innovation Laboratory, Center for Cancer Research, National Cancer Institute, Frederick, MD, USA.
Source: The Journal of pathology [J Pathol] 2023 Jul; Vol. 260 (3), pp. 276-288. Date of Electronic Publication: 2023 Apr 27.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: John Wiley And Sons Country of Publication: England NLM ID: 0204634 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1096-9896 (Electronic) Linking ISSN: 00223417 NLM ISO Abbreviation: J Pathol Subsets: MEDLINE
Database: MEDLINE Ultimate
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Description
ISSN:1096-9896
DOI:10.1002/path.6081