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The BCKDH kinase inhibitor BT2 promotes BCAA disposal and mitochondrial proton leak in both insulin-sensitive and insulin-resistant C2C12 myotubes.

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Title: The BCKDH kinase inhibitor BT2 promotes BCAA disposal and mitochondrial proton leak in both insulin-sensitive and insulin-resistant C2C12 myotubes.
Authors: Rivera CN; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., Smith CE; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., Draper LV; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., Kee ME; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., Cook NE; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., McGovern MR; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA., Watne RM; Department of Chemistry, High Point University, High Point, North Carolina, USA., Wommack AJ; Department of Chemistry, High Point University, High Point, North Carolina, USA., Vaughan RA; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.
Source: Journal of cellular biochemistry [J Cell Biochem] 2024 Mar; Vol. 125 (3), pp. e30520. Date of Electronic Publication: 2024 Jan 16.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Wiley-Liss Country of Publication: United States NLM ID: 8205768 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1097-4644 (Electronic) Linking ISSN: 07302312 NLM ISO Abbreviation: J Cell Biochem Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: The BCKDH kinase inhibitor BT2 promotes BCAA disposal and mitochondrial proton leak in both insulin-sensitive and insulin-resistant C2C12 myotubes.
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  Data: <searchLink fieldCode="AU" term="%22Rivera+CN%22">Rivera CN</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Smith+CE%22">Smith CE</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Draper+LV%22">Draper LV</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Kee+ME%22">Kee ME</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Cook+NE%22">Cook NE</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22McGovern+MR%22">McGovern MR</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Watne+RM%22">Watne RM</searchLink>; Department of Chemistry, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Wommack+AJ%22">Wommack AJ</searchLink>; Department of Chemistry, High Point University, High Point, North Carolina, USA.<br /><searchLink fieldCode="AU" term="%22Vaughan+RA%22">Vaughan RA</searchLink>; Department of Health and Human Performance, High Point University, High Point, North Carolina, USA.
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  Data: <searchLink fieldCode="JN" term="%228205768%22">Journal of cellular biochemistry</searchLink> [J Cell Biochem] 2024 Mar; Vol. 125 (3), pp. e30520. <i>Date of Electronic Publication: </i>2024 Jan 16.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Wiley-Liss%22">Wiley-Liss </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>8205768 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1097-4644 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2207302312%22">07302312 </searchLink><i>NLM ISO Abbreviation: </i>J Cell Biochem <i>Subsets: </i>MEDLINE
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        Value: 10.1002/jcb.30520
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      – Code: eng
        Text: English
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        StartPage: e30520
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      – TitleFull: The BCKDH kinase inhibitor BT2 promotes BCAA disposal and mitochondrial proton leak in both insulin-sensitive and insulin-resistant C2C12 myotubes.
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              Text: 2024 Mar
              Type: published
              Y: 2024
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