Depletion of SAM leading to loss of heterochromatin drives muscle stem cell ageing.

Saved in:
Bibliographic Details
Title: Depletion of SAM leading to loss of heterochromatin drives muscle stem cell ageing.
Authors: Kang J; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Benjamin DI; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Kim S; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Salvi JS; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Dhaliwal G; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Lam R; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Goshayeshi A; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Brett JO; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Liu L; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA., Rando TA; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Neurology Service, Veterans Affairs Palo Alto Healthcare System, Palo Alto, CA, USA. trando@mednet.ucla.edu.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA. trando@mednet.ucla.edu.
Source: Nature metabolism [Nat Metab] 2024 Jan; Vol. 6 (1), pp. 153-168. Date of Electronic Publication: 2024 Jan 19.
Publication Type: Journal Article
Journal Info: Publisher: Springer Nature Country of Publication: Germany NLM ID: 101736592 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2522-5812 (Electronic) Linking ISSN: 25225812 NLM ISO Abbreviation: Nat Metab Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2522-5812
DOI:10.1038/s42255-023-00955-z