Depletion of SAM leading to loss of heterochromatin drives muscle stem cell ageing.

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Title: Depletion of SAM leading to loss of heterochromatin drives muscle stem cell ageing.
Authors: Kang J; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Benjamin DI; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Kim S; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Salvi JS; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Dhaliwal G; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Lam R; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Goshayeshi A; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Brett JO; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA., Liu L; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA., Rando TA; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Neurology Service, Veterans Affairs Palo Alto Healthcare System, Palo Alto, CA, USA. trando@mednet.ucla.edu.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA. trando@mednet.ucla.edu.
Source: Nature metabolism [Nat Metab] 2024 Jan; Vol. 6 (1), pp. 153-168. Date of Electronic Publication: 2024 Jan 19.
Publication Type: Journal Article
Journal Info: Publisher: Springer Nature Country of Publication: Germany NLM ID: 101736592 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2522-5812 (Electronic) Linking ISSN: 25225812 NLM ISO Abbreviation: Nat Metab Subsets: MEDLINE
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  Data: <searchLink fieldCode="AU" term="%22Kang+J%22">Kang J</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Benjamin+DI%22">Benjamin DI</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Kim+S%22">Kim S</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Salvi+JS%22">Salvi JS</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Dhaliwal+G%22">Dhaliwal G</searchLink>; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Lam+R%22">Lam R</searchLink>; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Goshayeshi+A%22">Goshayeshi A</searchLink>; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Brett+JO%22">Brett JO</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.<br /><searchLink fieldCode="AU" term="%22Liu+L%22">Liu L</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA.<br /><searchLink fieldCode="AU" term="%22Rando+TA%22">Rando TA</searchLink>; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Paul F. Glenn Laboratories for the Biology of Aging, Stanford University School of Medicine, Stanford, CA, USA. trando@mednet.ucla.edu.; Neurology Service, Veterans Affairs Palo Alto Healthcare System, Palo Alto, CA, USA. trando@mednet.ucla.edu.; Department of Neurology and Broad Stem Cell Research Center, University of California, Los Angeles, Los Angeles, CA, USA. trando@mednet.ucla.edu.
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