Regulatory T cells use heparanase to access IL-2 bound to extracellular matrix in inflamed tissue.
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| Title: | Regulatory T cells use heparanase to access IL-2 bound to extracellular matrix in inflamed tissue. |
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| Authors: | Martinez HA; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Koliesnik I; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Kaber G; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Reid JK; Department of Clinical Neurosciences, Cumming School of Medicine, University of Calgary, Calgary, AB, Canada.; Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, Calgary, Canada.; Snyder Institute for Chronic Diseases, Cumming School of Medicine, University of Calgary, Calgary, Canada., Nagy N; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Barlow G; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Falk BA; Matrix Biology Program, Benaroya Research Institute, Seattle, WA, USA., Medina CO; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Hargil A; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Zihsler S; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Vlodavsky I; Technion Integrated Cancer Center, Technion, Haifa, Israel., Li JP; Department of Medical Biochemistry and Microbiology, Uppsala University, Uppsala, Sweden., Pérez-Cruz M; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Tang SW; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Meyer EH; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Wrenshall LE; Department of Neuroscience, Cell Biology, and Physiology, Boonshoft School of Medicine, Wright State University, Dayton, OH, USA., Lord JD; Translational Research Program, Benaroya Research Institute, Seattle, WA, USA., Garcia KC; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA., Palmer TD; Department of Neurosurgery, Stanford University School of Medicine, Stanford, CA, USA., Steinman L; Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA., Nepom GT; Immune Tolerance Network, Benaroya Research Institute, Seattle, WA, USA., Wight TN; Matrix Biology Program, Benaroya Research Institute, Seattle, WA, USA., Bollyky PL; Department of Medicine, Stanford University School of Medicine, Stanford, CA, USA., Kuipers HF; Department of Clinical Neurosciences, Cumming School of Medicine, University of Calgary, Calgary, AB, Canada. hedwich.kuipers@ucalgary.ca.; Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, Calgary, Canada. hedwich.kuipers@ucalgary.ca.; Snyder Institute for Chronic Diseases, Cumming School of Medicine, University of Calgary, Calgary, Canada. hedwich.kuipers@ucalgary.ca.; Department of Cell Biology and Anatomy, Cumming School of Medicine, University of Calgary, Calgary, Canada. hedwich.kuipers@ucalgary.ca. |
| Source: | Nature communications [Nat Commun] 2024 Feb 20; Vol. 15 (1), pp. 1564. Date of Electronic Publication: 2024 Feb 20. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 2041-1723 |
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| DOI: | 10.1038/s41467-024-45012-9 |
