Cleavage of Hsp70.1 causes lysosomal cell death under stress conditions.

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Bibliographic Details
Title: Cleavage of Hsp70.1 causes lysosomal cell death under stress conditions.
Authors: Yamashima T; Department of Psychiatry and Behavioral Science, Kanazawa University Graduate School of Medical Sciences, Kanazawa, Japan.; Department of Gastroenterology, Kanazawa University Graduate School of Medical Sciences, Kanazawa, Japan., Mochly-Rosen D; Department of Chemical and Systems Biology, Stanford University School of Medicine, Stanford, CA, United States., Wakatsuki S; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, United States., Mizukoshi E; Department of Gastroenterology, Kanazawa University Graduate School of Medical Sciences, Kanazawa, Japan., Seike T; Department of Gastroenterology, Kanazawa University Graduate School of Medical Sciences, Kanazawa, Japan.; Department of Chemical and Systems Biology, Stanford University School of Medicine, Stanford, CA, United States., Larus IM; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, United States., Chen CH; Department of Chemical and Systems Biology, Stanford University School of Medicine, Stanford, CA, United States., Takemura M; Laboratory of Gene Function, Research Institute for Bioresources and Biotechnology, Ishikawa Prefectural University, Nonoichi, Japan., Saito H; Division of Collaborative Research and Development, Exploratory Oncology Research and Clinical Trial Center, National Cancer Center, Kashiwa, Japan., Ohashi A; Division of Collaborative Research and Development, Exploratory Oncology Research and Clinical Trial Center, National Cancer Center, Kashiwa, Japan.
Source: Frontiers in molecular biosciences [Front Mol Biosci] 2024 May 27; Vol. 11, pp. 1378656. Date of Electronic Publication: 2024 May 27 (Print Publication: 2024).
Publication Type: Journal Article; Review
Journal Info: Publisher: Frontiers Media S.A Country of Publication: Switzerland NLM ID: 101653173 Publication Model: eCollection Cited Medium: Print ISSN: 2296-889X (Print) Linking ISSN: 2296889X NLM ISO Abbreviation: Front Mol Biosci Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2296-889X
DOI:10.3389/fmolb.2024.1378656