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Activation of the Keap1/Nrf2 pathway suppresses mitochondrial dysfunction, oxidative stress, and motor phenotypes in C9orf72 ALS/FTD models.

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Title: Activation of the Keap1/Nrf2 pathway suppresses mitochondrial dysfunction, oxidative stress, and motor phenotypes in C9orf72 ALS/FTD models.
Authors: Au WH; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK., Miller-Fleming L; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK., Sanchez-Martinez A; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK., Lee JA; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK., Twyning MJ; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK., Prag HA; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.; Department of Medicine, University of Cambridge, Cambridge, UK., Raik L; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK., Allen SP; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK., Shaw PJ; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.; NIHR Sheffield Biomedical Research Centre, Sheffield Teaching Hospitals NHS Foundation Trust, Sheffield, UK., Ferraiuolo L; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK., Mortiboys H; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK., Whitworth AJ; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK a.whitworth@mrc-mbu.cam.ac.uk.
Source: Life science alliance [Life Sci Alliance] 2024 Jun 21; Vol. 7 (9). Date of Electronic Publication: 2024 Jun 21 (Print Publication: 2024).
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Life Science Alliance, LLC Country of Publication: United States NLM ID: 101728869 Publication Model: Electronic-Print Cited Medium: Internet ISSN: 2575-1077 (Electronic) Linking ISSN: 25751077 NLM ISO Abbreviation: Life Sci Alliance Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Activation of the Keap1/Nrf2 pathway suppresses mitochondrial dysfunction, oxidative stress, and motor phenotypes in C9orf72 ALS/FTD models.
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  Data: <searchLink fieldCode="AU" term="%22Au+WH%22">Au WH</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Miller-Fleming+L%22">Miller-Fleming L</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Sanchez-Martinez+A%22">Sanchez-Martinez A</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Lee+JA%22">Lee JA</searchLink>; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.<br /><searchLink fieldCode="AU" term="%22Twyning+MJ%22">Twyning MJ</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Prag+HA%22">Prag HA</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.; Department of Medicine, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Raik+L%22">Raik L</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Allen+SP%22">Allen SP</searchLink>; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.<br /><searchLink fieldCode="AU" term="%22Shaw+PJ%22">Shaw PJ</searchLink>; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.; NIHR Sheffield Biomedical Research Centre, Sheffield Teaching Hospitals NHS Foundation Trust, Sheffield, UK.<br /><searchLink fieldCode="AU" term="%22Ferraiuolo+L%22">Ferraiuolo L</searchLink>; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.<br /><searchLink fieldCode="AU" term="%22Mortiboys+H%22">Mortiboys H</searchLink>; Sheffield Institute for Translational Neuroscience (SITraN), School of Medicine and Population Health, University of Sheffield, Sheffield, UK.<br /><searchLink fieldCode="AU" term="%22Whitworth+AJ%22">Whitworth AJ</searchLink>; MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge, UK a.whitworth@mrc-mbu.cam.ac.uk.
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  Data: <searchLink fieldCode="JN" term="%22101728869%22">Life science alliance</searchLink> [Life Sci Alliance] 2024 Jun 21; Vol. 7 (9). <i>Date of Electronic Publication: </i>2024 Jun 21 (<i>Print Publication: </i>2024).
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