Butyrate and propionate are microbial danger signals that activate the NLRP3 inflammasome in human macrophages upon TLR stimulation.

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Title: Butyrate and propionate are microbial danger signals that activate the NLRP3 inflammasome in human macrophages upon TLR stimulation.
Authors: Wang W; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany; Wellcome-Wolfson Institute for Experimental Medicine, Queen's University Belfast, Belfast BT9 7BL, UK., Dernst A; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Martin B; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Lorenzi L; Josep Carreras Leukemia Research Institute (IJC), 08916 Badalona, Barcelona, Spain., Cadefau-Fabregat M; Josep Carreras Leukemia Research Institute (IJC), 08916 Badalona, Barcelona, Spain., Phulphagar K; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Wagener A; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Budden C; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Stair N; Institute for Genetics, CECAD Research Center, University of Cologne, 50931 Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging-associated Diseases (CECAD), University of Cologne, 50931 Cologne, Germany., Wagner T; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Färber H; Institute for Hygiene and Public Health, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Jaensch A; Institute for Hygiene and Public Health, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Stahl R; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Duthie F; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Schmidt SV; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Coll RC; Wellcome-Wolfson Institute for Experimental Medicine, Queen's University Belfast, Belfast BT9 7BL, UK., Meissner F; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany., Cuartero S; Josep Carreras Leukemia Research Institute (IJC), 08916 Badalona, Barcelona, Spain., Latz E; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany; German Center for Neurodegenerative Diseases, 53127 Bonn, Germany; Department of Infectious Diseases & Immunology, UMass Medical School, Worcester, MA 01605, USA; Deutsches Rheuma Forschungszentrum Berlin (DRFZ), 10117 Berlin, Germany. Electronic address: eicke.latz@drfz.de., Mangan MSJ; Institute of Innate Immunity, University Hospital Bonn, University of Bonn, 53127 Bonn, Germany; German Center for Neurodegenerative Diseases, 53127 Bonn, Germany. Electronic address: matthew.mangan@ukbonn.de.
Source: Cell reports [Cell Rep] 2024 Sep 24; Vol. 43 (9), pp. 114736. Date of Electronic Publication: 2024 Sep 13.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Cell Press Country of Publication: United States NLM ID: 101573691 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2211-1247 (Electronic) NLM ISO Abbreviation: Cell Rep Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2211-1247
DOI:10.1016/j.celrep.2024.114736